Santhosh Kumar

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Assistant Research Professor

Indiana State University

United States

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About

Santhosh Kumar is an Assistant Research Professor at Indiana University, USA. His research primarily focuses on the interactions between immune microenvironments and leukemic pathways, with recent publications exploring the IL-33/IL1RL1 axis, the effects of obesity-induced inflammation on clonal hematopoiesis, and novel therapeutic strategies for drug-resistant acute myeloid leukemia. Dr. Kumar's work aims to enhance understanding of hematopoietic homeostasis and its implications for leukemia treatment.

Recent Grants

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In vitro differentiation and characterization of CD34+ stem cells.

Open Date: 2016-10-01

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Articles (14)

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2025

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2021

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2020

Collaborators (6)

Baskar Ramdas

Associate Research Professor

University of Indianapolis

UNITED STATES

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Benjamin Ebert

Dana-Farber Cancer Institute

UNITED STATES

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Sophie Paczesny

Medical University of South Carolina

UNITED STATES

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Elliot Stieglitz

Associate Professor

UCSF Benioff Children's Hospital

UNITED STATES

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Chi Zhang

Associate Professor

Indiana University School of Medicine

UNITED STATES

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Mark Kelley

Professor of Pediatrics, Senior Leader in Biomedical Innovation and Partnership

Indiana University-Purdue University Indianapolis

UNITED STATES

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Santhosh Kumar

Research Interests

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About

Recent Grants

Articles (14)

ST2/IL-33 axis blockade inhibits regulatory T cell cytotoxicity towards CD8 T cells in the leukemic niche

Dual targeting of tumoral cells and immune microenvironment by blocking the IL-33/IL1RL1 pathway

Alkynyl nicotinamides show antileukemic activity in drug-resistant acute myeloid leukemia

The impact of obesity-induced inflammation on clonal hematopoiesis

Obesity-induced inflammation exacerbates clonal hematopoiesis

Potential clinical use of azacitidine and MEK inhibitor combination therapy in PTPN11-mutated juvenile myelomonocytic leukemia.

Loss of Dnmt3a impairs hematopoietic homeostasis and myeloid cell skewing via the PI3Kinase pathway

Inhibition of BTK and PI3Kδ impairs the development of human JMML stem and progenitor cells.

Combined heterozygosity of FLT3 ITD, TET2, and DNMT3A results in aggressive leukemia

Il-1r1 drives leukemogenesis induced by Tet2 loss

Obesity-Induced Inflammation Co-Operates with Clonal Hematopoiesis of Indeterminate Potential (CHIP) Mutants to Promote Leukemia Development and Cardiovascular Disease

Targeting M2-Tumor Associated Macrophages By Arginase-1 and PD-L1 in Regulating Juvenile Myelomonocytic Leukemia (JMML) Development and Relapse

Staphylococcus aureus grown in anaerobic conditions exhibits elevated glutamine biosynthesis and biofilm units

Exploiting the proteasome to overcome RAS and WT1 mutation mediated resistance to FLT-3 inhibition in acute myeloid leukemia

Collaborators (6)

Baskar Ramdas

Benjamin Ebert

Sophie Paczesny

Elliot Stieglitz

Chi Zhang

Mark Kelley

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