Sarah Latchney

Assistant Professor

St. Mary's College of Maryland

United States

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About

Sarah Latchney is an Assistant Professor at St. Mary's College of Maryland, USA. Her research focuses on neurobiology, particularly the effects of genetic changes and environmental factors on neurogenesis and brain function. Recent articles authored by her investigate topics such as DNA methylation patterns in response to maternal upbringing, the impact of specific pollutants on microglial function, and the role of B cells in brain recovery following injury.

Articles (9)

Year:

2025

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2023

Year:

2023

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2022

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2022

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2021

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2021

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2020

Year:

2020

Collaborators (5)

Theodore Garland

University of California, Riverside

UNITED STATES

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Ania Majewska

Professor

University of Rochester

UNITED STATES

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Mark Paul Goldberg

Associate VP Strategic Research Initiatives / Professor

University of Texas Health Science Center at San Antonio

UNITED STATES

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Rebecca Lowery

University of Rochester Medical Center

UNITED STATES

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H. Mark Kenney

University of Rochester Medical Center

UNITED STATES

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Sarah Latchney

Research Interests

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Articles (9)

Glial changes in the dentate gyrus of neuronal-specific PTEN knockout mice correlate with changes in cell proliferation.

Maternal upbringing and selective breeding for voluntary exercise behavior modify patterns of DNA methylation and expression of genes in the mouse brain

Neuronal deletion of phosphatase and tensin homolog in mice results in spatial dysregulation of adult hippocampal neurogenesis

DNA Methylation Analysis of Imprinted Genes in the Cortex and Hippocampus of Cross-Fostered Mice Selectively Bred for Increased Voluntary Wheel-Running

Gestational and lactational exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin primes cortical microglia to tissue injury.

Characterization of the temporal, cell-specific and interferon-inducible patterns of indoleamine 2,3 dioxygenase 1 (IDO1) expression in the human placenta across gestation

Persistent organic pollutants at the synapse: Shared phenotypes and converging mechanisms of developmental neurotoxicity

Acute 2,3,7,8-Tetrachlorodibenzo-p-dioxin exposure in adult mice does not alter the morphology or inflammatory response of cortical microglia.

B cells migrate into remote brain areas and support neurogenesis and functional recovery after focal stroke in mice.

Collaborators (5)

Theodore Garland

Ania Majewska

Mark Paul Goldberg

Rebecca Lowery

H. Mark Kenney

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