Sasha Shafikhani

Professor

Rush University Medical Center

United States

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About

Dr. Sasha Shafikhani is a Professor at Rush University Medical Center in the United States. His research focuses on skin tissue engineering, wound healing, and the interactions between bacterial infections and immune responses. Recent publications include studies on innovative materials for wound management and the mechanisms of Pseudomonas aeruginosa cytotoxicity.

Articles (15)

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2025

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2025

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2022

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2021

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2021

Collaborators (2)

Dawn Wetzel

Assistant Professor

University of Texas Southwestern Medical Center

UNITED STATES

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K A Foyez Mahmud

Rush University Medical Center

UNITED STATES

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Sasha Shafikhani

Research Interests

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Articles (15)

Engineering of a Bilayer Film Made From Decellularized Amniotic Membrane and Emulsion for Skin Tissue Engineering Applications

Engineering of a biomechanically and biologically improved amniotic membrane using nanofibrous bacterial cellulose for healing of corneal defects

Reduced Bioactive Microbial Products (Pathogen-Associated Molecular Patterns) Contribute to Dysregulated Immune Responses and Impaired Healing in Infected Wounds in Mice with Diabetes

Engineering of a bilayer antibacterial wound dressing from bovine pericardium and electrospun chitosan/PVA/antibiotics for infectious skin wounds management: An in vitro and in vivo study.

Optimization of an Affordable and Efficient Skin Allograft Composite with Excellent Biomechanical and Biological Properties Suitable for the Regeneration of Deep Skin Wounds: A Preclinical Study.

An Updated Review of Hypertrophic Scarring

Pseudomonas aeruginosa Cytotoxins: Mechanisms of Cytotoxicity and Impact on Inflammatory Responses

Pseudomonas aeruginosa: Infections, Animal Modeling, and Therapeutics

CrkII/Abl phosphorylation cascade is critical for NLRC4 inflammasome activity and is blocked by Pseudomonas aeruginosa ExoT

Presence of CrkI-containing microvesicles in squamous cell carcinomas could have ramifications on tumor biology and cancer therapeutics.

IL-10 Dysregulation Underlies Chemokine Insufficiency, Delayed Macrophage Response, and Impaired Healing in Diabetic Wounds

Overriding impaired FPR chemotaxis signaling in diabetic neutrophil stimulates infection control in murine diabetic wound.

Therapeutic evaluation of immunomodulators in reducing surgical wound infection

Impact of preconditioned diabetic stem cells and photobiomodulation on quantity and degranulation of mast cells in a delayed healing wound simulation in type one diabetic rats.

Therapeutic assessment of N-formyl-methionyl-leucyl-phenylalanine (fMLP) in reducing periprosthetic joint infection.

Collaborators (2)

Dawn Wetzel

K A Foyez Mahmud

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