Sathish K. R. Padi

Assistant Professor (In Residence)

University of Connecticut Health Center

United States

Research Interests

Explore related searches

Professors in United States Professors in Cancer Biology Cancer Biology professors in United States

Contact this professor

Get a free first email built on one of their papers, ready for you to edit and send yourself.

About

Dr. Sathish K. R. Padi is an Assistant Professor (In Residence) at UConn Health Center in the United States. His research focuses on the molecular mechanisms of phosphatase regulation, particularly the role of PP2A and its impact on cancer biology. Recent publications highlight his work utilizing cryo-electron microscopy to elucidate substrate recruitment and signaling pathways involved in leukemia and tumor plasticity.

Articles (10)

Year:

2025

Year:

2025

Year:

2025

Year:

2024

Year:

2021

Year:

2021

Year:

2021

Year:

2020

Year:

2020

Year:

2020

Collaborators (2)

Jakob Nilsson

Professor

University of Copenhagen

DENMARK

View details

JIN SONG

Associate Research Professor

University of Florida

UNITED STATES

View details

ApplyKite TwinLearns as you apply

Make your next move.

Prepare your application

Review drafts before you send them.

Sathish K. R. Padi

Research Interests

Explore related searches

Contact this professor

About

Articles (10)

Cryo-EM structures of PP2A:B55 with p107 and Eya3 define substrate recruitment.

Molecular mechanism of PP2A/B55α phosphatase inhibition by IER5.

Combining cryo-electron microscopy (cryo-EM) with orthogonal solution state methods to define the molecular basis of the phosphoprotein phosphatase family regulation and substrate specificity

Cryo-EM structures of PP2A:B55–FAM122A and PP2A:B55–ARPP19

The long noncoding RNA H19 regulates tumor plasticity in neuroendocrine prostate cancer.

"Root"ing for successful T-ALL treatment.

EDC3 phosphorylation regulates growth and invasion through controlling P‐body formation and dynamics

PIM Kinase Inhibitors Block the Growth of Primary T-cell Acute Lymphoblastic Leukemia: Resistance Pathways Identified by Network Modeling Analysis.

PIM protein kinases regulate the level of the long noncoding RNA H19 to control stem cell gene transcription and modulate tumor growth.

PIM Kinase Inhibitors Block the Growth of Primary T-cell Acute Lymphoblastic Leukemia: Resistance Pathways Identified by Network Modeling Analysis

Collaborators (2)

Jakob Nilsson

JIN SONG

Make your next move.