Satoshi Tanaka

Professor

Kyoto Pharmaceutical University

Japan

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About

Professor Satoshi Tanaka is a faculty member at Kyoto Pharmaceutical University in Japan. His research primarily focuses on mast cell biology, allergic responses, and the interaction of immune cells with sensory neurons. Recent publications include studies on the mechanisms of mast cell degranulation and the roles of various receptors in immune signaling.

Articles (11)

Year:

2024

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2024

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2024

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2024

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2024

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2023

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2023

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2021

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2020

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2020

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2020

Collaborators (4)

Chikara Kaito

JAPAN

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Paul Roche

National Cancer Institute

UNITED STATES

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Satoshi Ishii

Akita University Graduate School of Medicine

JAPAN

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Isao Matsuoka

Professor

Takasaki University of Health and Welfare

JAPAN

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Satoshi Tanaka

Research Interests

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Articles (11)

<i>Staphylococcus aureus</i> δ-Toxin Induces Ca<sup>2+</sup> Influx-Independent Degranulation of Murine Cultured Mast Cells

IL‐33 Sensitizes Mast Cells to PIEZO1 Stimulation Leading to Degranulation

MrgprA3<sup>+</sup> Primary Sensory Neurons Mediate Acute Allergic Itch Responses in Atopic Dermatitis Model Mice

Lipid-orchestrated paracrine circuit coordinates mast cell maturation and anaphylaxis through functional interaction with fibroblasts

Suppression of Mast Cell Activation by GPR35: GPR35 Is a Primary Target of Disodium Cromoglycate

ATP and its metabolite adenosine cooperatively upregulate the antigen-presenting molecules on dendritic cells leading to IFN-γ production by T cells

Mast cells link immune sensing to antigen-avoidance behaviour

Roles of IgE and Histamine in Mast Cell Maturation

Ligation of MHC Class II Induces PKC-Dependent Clathrin-Mediated Endocytosis of MHC Class II.

Extracellular ATP Augments Antigen-Induced Murine Mast Cell Degranulation and Allergic Responses via P2X4 Receptor Activation

Phenotypic and Functional Diversity of Mast Cells

Collaborators (4)

Chikara Kaito

Paul Roche

Satoshi Ishii

Isao Matsuoka

Make your next move.

Satoshi Tanaka

Research Interests

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Contact this professor

About

Articles (11)

&lt;i&gt;Staphylococcus aureus&lt;/i&gt; δ-Toxin Induces Ca&lt;sup&gt;2+&lt;/sup&gt; Influx-Independent Degranulation of Murine Cultured Mast Cells

IL‐33 Sensitizes Mast Cells to PIEZO1 Stimulation Leading to Degranulation

MrgprA3&lt;sup&gt;+&lt;/sup&gt; Primary Sensory Neurons Mediate Acute Allergic Itch Responses in Atopic Dermatitis Model Mice

Lipid-orchestrated paracrine circuit coordinates mast cell maturation and anaphylaxis through functional interaction with fibroblasts

Suppression of Mast Cell Activation by GPR35: GPR35 Is a Primary Target of Disodium Cromoglycate

ATP and its metabolite adenosine cooperatively upregulate the antigen-presenting molecules on dendritic cells leading to IFN-γ production by T cells

Mast cells link immune sensing to antigen-avoidance behaviour

Roles of IgE and Histamine in Mast Cell Maturation

Ligation of MHC Class II Induces PKC-Dependent Clathrin-Mediated Endocytosis of MHC Class II.

Extracellular ATP Augments Antigen-Induced Murine Mast Cell Degranulation and Allergic Responses via P2X4 Receptor Activation

Phenotypic and Functional Diversity of Mast Cells

Collaborators (4)

Chikara Kaito

Paul Roche

Satoshi Ishii

Isao Matsuoka

Make your next move.

<i>Staphylococcus aureus</i> δ-Toxin Induces Ca<sup>2+</sup> Influx-Independent Degranulation of Murine Cultured Mast Cells

MrgprA3<sup>+</sup> Primary Sensory Neurons Mediate Acute Allergic Itch Responses in Atopic Dermatitis Model Mice