Sean wilson

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Associate Professor

Loma Linda University School of Medicine

United States

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Sean Wilson is an Associate Professor at Loma Linda University School of Medicine in the United States. His research areas include the physiological and pathological roles of calcium channels in blood pressure regulation, the effects of maternal exercise on offspring development, and innovative approaches to vascular graft performance. He has recently published articles on team learning in medical training, the impact of exercise on chronic conditions, and methodologies for analyzing vascular myocyte activity.

Recent Grants

Grant: Close

Maternal Hypoxemia and Oxidative Stress in the Fetus, Newborn, and Adult

Open Date: 2019-03-01

Close Date: 2021-02-28

Grant: Close

Intrauterine chronic hypoxia and ryanodine receptors in fetal pulmonary arteries

Open Date: 2012-06-01

Close Date: 2015-05-31

Grant: Close

MRI: Acquisition of a multiphoton confocal microscope for research, training and teaching

Open Date: 2009-09-15

Close Date: 2014-08-31

Grant: Close

Center of Research Excellence in Natural Products Neuro*

Open Date: 2006-09-04

Close Date: 2012-06-30

Grant: Close

Acquisition of a Confocal Microscope for Multidisciplinary Research, Research Training and Teaching

Open Date: 2006-08-01

Close Date: 2010-07-31

Articles (17)

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UNITED STATES

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Xiang-Qun Hu

Associate Professor

Loma Linda University

UNITED STATES

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RUI SONG

Loma Linda University

UNITED STATES

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Montri Wongworawat

Loma Linda University School of Medicine

UNITED STATES

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Kin-Hing Lau

Research Professor

Loma Linda University School of Medicine

UNITED STATES

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Sean wilson

Research Interests

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About

Recent Grants

Articles (17)

Evaluating a novel team learning approach for integrating drugs in sports education in preclinical medical training

H2S Increases Blood Pressure via Activation of L-Type Calcium Channels with Mediation by HS• Generated from Reactions with Oxyhemoglobin

Impact of Maternal Exercise on Mice Offspring Development, Pulmonary Hypertension, and Vascular Remodeling in Chronic Hypoxia

Long-term hypoxia modulates depolarization activation of BKCa currents in fetal sheep middle cerebral arterial myocytes

Improved Workflow for Analysis of Vascular Myocyte Time-Series and Line-Scan Ca2+ Imaging Datasets

A Biomimetic Approach Utilizing Pulsatile Perfusion Generates Contractile Vascular Grafts.

Can Maternal Exercise Prevent High-Altitude Pulmonary Hypertension in Children?

Editorial: The role of pericytes in physiology and pathophysiology

Middle school cycling program is associated with improved mental health and wellbeing in adolescents during COVID-19

Defective bone repletion in aged Balb/cBy mice was caused by impaired osteoblastic differentiation

Calcium released by osteoclastic resorption stimulates autocrine/paracrine activities in local osteogenic cells to promote coupled bone formation

Ryanodine receptor subtypes regulate Ca2+ sparks/spontaneous transient outward currents and myogenic tone of uterine arteries in pregnancy

The Effects of Insulin-Like Growth Factor I and BTP-2 on Acute Lung Injury

MicroRNA-210 Mediates Hypoxia-Induced Repression of Spontaneous Transient Outward Currents in Sheep Uterine Arteries During Gestation.

Gestational long-term hypoxia induces metabolomic reprogramming and phenotypic transformations in fetal sheep pulmonary arteries.

Gestational Hypoxia Inhibits Pregnancy-Induced Upregulation of Ca<sup>2+</sup> Sparks and Spontaneous Transient Outward Currents in Uterine Arteries Via Heightened Endoplasmic Reticulum/Oxidative Stress.

IGF-1 Deficiency Rescue and Intracellular Calcium Blockade Improves Survival and Corresponding Mechanisms in a Mouse Model of Acute Kidney Injury.

Collaborators (10)

Mary Kearns-Jonker

DaLiao Xiao

Samiksha Wasnik

Abu Ahmed

Lubo Zhang

David J. Baylink

Xiang-Qun Hu

RUI SONG

Montri Wongworawat

Kin-Hing Lau

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