Seiji Watanabe

Lecturer

Japan

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Articles (13)

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2024

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2024

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2024

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2023

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2023

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2023

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2023

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2021

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2021

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2021

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2020

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2020

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2020

Collaborators (6)

Kotaro Oiwa

Designated Assistant Professor

Nagoya University

JAPAN

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Norifumi Muraki

Institute for Molecular Science

JAPAN

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Yohei Okada

Professor

Aichi Medical University

JAPAN

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Yoshiaki Furukawa

JAPAN

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Yohei Iguchi

Assistant Professor

Nagoya University

JAPAN

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Koji Yamanaka

Professor

Nagoya University

JAPAN

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Seiji Watanabe

Research Interests

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Articles (13)

Genetic background variation impacts microglial heterogeneity and disease progression in amyotrophic lateral sclerosis model mice

Ebselen analogues delay disease onset and its course in fALS by on-target SOD-1 engagement

Dimethyl fumarate improves cognitive impairment and neuroinflammation in mice with Alzheimer’s disease

Monomerization of TDP-43 is a key determinant for inducing TDP-43 pathology in amyotrophic lateral sclerosis

Intrinsic structural vulnerability in the hydrophobic core induces species-specific aggregation of canine SOD1 with degenerative myelopathy–linked E40K mutation

Sigma-1 receptor maintains ATAD3A as a monomer to inhibit mitochondrial fragmentation at the mitochondria-associated membrane in amyotrophic lateral sclerosis.

Mitochondria-associated membrane collapse impairs TBK1-mediated proteostatic stress response in ALS.

Novel reporters of mitochondria‐associated membranes (MAM), MAMtrackers, demonstrate MAM disruption as a common pathological feature in amyotrophic lateral sclerosis

Microglial gene signature reveals loss of homeostatic microglia associated with neurodegeneration of Alzheimer’s disease

Novel reporters of mitochondria-associated membranes (MAM), MAMtrackers, demonstrate MAM disruption as a common pathological feature in amyotrophic lateral sclerosis.

Aggresome formation and liquid–liquid phase separation independently induce cytoplasmic aggregation of TAR DNA-binding protein 43

ALS-linked TDP-43M337V knock-in mice exhibit splicing deregulation without neurodegeneration

Novel Selenium-based compounds with therapeutic potential for SOD1-linked amyotrophic lateral sclerosis.

Collaborators (6)

Kotaro Oiwa

Norifumi Muraki

Yohei Okada

Yoshiaki Furukawa

Yohei Iguchi

Koji Yamanaka

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