Seung Goo Kang

Associate Professor

Changwon National University

South Korea

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About

Seung Goo Kang is an Associate Professor at Kangwon National University in South Korea. His research focuses on immunology and cell death mechanisms, particularly in the context of multiple myeloma and obesity. Recent publications include studies on the effects of lactoferrin-derived peptides and the role of immune cells in inflammatory processes. He explores innovative therapeutic approaches aimed at enhancing regulatory T cell differentiation and addressing metabolic dysregulation.

Articles (12)

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2025

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2025

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2023

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2021

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2021

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2021

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2021

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2021

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2020

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2020

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2020

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2020

Collaborators (9)

Wooseob Kim

Assistant professor

Yonsei University College of Medicine

SOUTH KOREA

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John Teijaro

Professor

Scripps Research Institute

UNITED STATES

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Hyun-Jeong Ko

Professor

Changwon National University

SOUTH KOREA

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Jung-Shin Lee

Assistant Professor

Changwon National University

SOUTH KOREA

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Hyung‐seung Jin

SOUTH KOREA

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Eunsong Lee

Professor

Changwon National University

SOUTH KOREA

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Hojun Kim

SOUTH KOREA

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Seok-Rae Park

Professor

Konyang University - Daejeon Medical Campus

SOUTH KOREA

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Gilyoung Lee

Changwon National University

SOUTH KOREA

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Seung Goo Kang

Research Interests

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Articles (12)

Lactoferrin-Derived Peptide Chimera Induces Caspase-Independent Cell Death in Multiple Myeloma

Bacterial components‐driven intrahepatic CXCR5hi B cells are important population for MASH progression through inducing inflammation

Combined Treatment With TGF-β1, Retinoic Acid, and Lactoferrin Robustly Generate Inducible Tregs (iTregs) Against High Affinity Ligand

Lactoferrin Potentiates Inducible Regulatory T Cell Differentiation through TGF-β Receptor III Binding and Activation of Membrane-Bound TGF-β

APB-F1, a long-acting feline granulocyte colony-stimulating factor fusion protein, created by exploiting FL335, a chimeric Fab specific for feline serum albumin

Effects of Live and Pasteurized Forms of Akkermansia from the Human Gut on Obesity and Metabolic Dysregulation

NLRP3 Triggers Attenuate Lipocalin-2 Expression Independent with Inflammasome Activation

IFNAR1 signaling in NK cells promotes persistent virus infection

GSK3 Restrains Germinal Center B Cells to Form Plasma Cells.

Activation of the Legionella pneumophila LegK7 Effector Kinase by the Host MOB1 Protein.

Lactoferrin Induces Tolerogenic Bone Marrow-Derived Dendritic Cells.

Cathelicidin-Related Antimicrobial Peptide Regulates CD73 Expression in Mouse Th17 Cells via p38

Collaborators (9)

Wooseob Kim

John Teijaro

Hyun-Jeong Ko

Jung-Shin Lee

Hyung‐seung Jin

Eunsong Lee

Hojun Kim

Seok-Rae Park

Gilyoung Lee

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