Shimpei Gotoh

Professor

Kyoto University

Japan

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About

Shimpei Gotoh is a Professor at Kyoto University in Japan. His research primarily focuses on the genetics and treatment of respiratory diseases, including primary ciliary dyskinesia and interstitial lung disease. He has contributed to the understanding of SARS-CoV-2 propagation and lung regenerative medicine through his recent publications.

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2024

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2020

Collaborators (7)

Shotaro MAEHANA

Kitasato University

JAPAN

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Kazuo Takayama

Junior Associate Professor

Kyoto University

JAPAN

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Ryuta Mikawa

Project Scientist, Assistant Professor

Kyoto University

JAPAN

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James Hagood

Professor

University of North Carolina at Chapel Hill School of Medicine

UNITED STATES

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Naoyuki Sone

Program-Specific Research Center Assistant Professor

Kyoto University

JAPAN

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Ryotaro Hashizume

Assistant Professor

Mie university

JAPAN

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Masato Suzuki

National Institute of Infectious Diseases

JAPAN

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Shimpei Gotoh

Research Interests

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About

Articles (20)

Micro-patterned culture of iPSC-derived alveolar and airway cells distinguishes SARS-CoV-2 variants.

A 3000-year-old founder variant in the DRC1 gene causes primary ciliary dyskinesia in Japan and Korea

Corrigendum: ACE2 knockout hinders SARS-CoV-2 propagation in iPS cell-derived airway and alveolar epithelial cells

Past and future of alveolar organoids for lung regenerative medicine

Practical guide for the diagnosis and management of primary ciliary dyskinesia

Screening of factors inducing alveolar type 1 epithelial cells using human pluripotent stem cells

Two Pediatric Cases of Primary Ciliary Dyskinesia Caused by Loss‐of‐Function Variants in Oral‐Facial‐Digital Syndrome Gene, <i>OFD1</i>

Contemporary and emerging technologies for research in children's rare and interstitial lung disease

ACE2 knockout hinders SARS-CoV-2 propagation in iPS cell-derived airway and alveolar epithelial cells

Cryptotanshinone is a candidate therapeutic agent for interstitial lung disease associated with a BRICHOS-domain mutation of SFTPC

Analysis of the clinical features of Japanese patients with primary ciliary dyskinesia

Disease modeling of pulmonary fibrosis using human pluripotent stem cell-derived alveolar organoids

Modeling of lung phenotype of Hermansky–Pudlak syndrome type I using patient-specific iPSCs

The importance of central airway dilatation in patients with bronchiolitis obliterans

Core-shell hydrogel microfiber-expanded pluripotent stem cell-derived lung progenitors applicable to lung reconstruction in vivo

Multicellular modeling of ciliopathy by combining iPS cells and microfluidic airway-on-a-chip technology

Hydroxypropyl Cyclodextrin Improves Amiodarone-induced Aberrant Lipid Homeostasis of Alveolar Cells

A pediatric case of primary ciliary dyskinesia caused by novel copy number variation in PIH1D3

Directed induction of alveolar type I cells derived from pluripotent stem cells via Wnt signaling inhibition

Copy number variation in DRC1 is the major cause of primary ciliary dyskinesia in the Japanese population.

Collaborators (7)

Shotaro MAEHANA

Kazuo Takayama

Ryuta Mikawa

James Hagood

Naoyuki Sone

Ryotaro Hashizume

Masato Suzuki

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