Shinji Takai

Professor

Osaka Medical and Pharmaceutical University

Japan

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About

Shinji Takai is a Professor at Osaka Medical and Pharmaceutical University in Japan. His research focuses on cardiovascular health, renal function, inflammation, and the therapeutic potential of various proteins and genes in related diseases. Recent studies include the beneficial effects of chymase inhibition in cardiac function, the role of erythropoietin in insulin resistance, and the impact of SPARC on endothelial inflammation and kidney injury.

Articles (10)

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2025

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2025

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2025

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2025

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2025

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2024

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2024

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2024

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2024

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2024

Collaborators (5)

Yoshimasa Tanaka

Director and Professor

Nagasaki University

JAPAN

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Hideki Tomiyama

Osaka Medical and Pharmaceutical University

JAPAN

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Hidehiro Oku

Osaka Medical and Pharmaceutical University

JAPAN

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Shinji Takai

Professor

Osaka Medical College

JAPAN

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TETSUO NAKATA

emeritus professor

Kyoto Pharmaceutical University

JAPAN

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Shinji Takai

Research Interests

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Articles (10)

Beneficial Effects of Chymase Inhibition on Cardiac Diastolic Function and Remodeling Induced by Chronic Angiotensin II Stimulation

Erythropoietin Attenuates Insulin Resistance and Renal Inflammation in High-Sucrose-Treated Rats

Suppressing SPARC gene with siRNA exerts therapeutic effects and inhibits MMP-2/9 and ADAMTS1 overexpression in a murine model of ischemia/reperfusion-induced acute kidney injury

Secreted Protein Acidic and Rich in Cysteine (SPARC) Induced by the Renin–Angiotensin System Causes Endothelial Inflammation in the Early Stages of Hypertensive Vascular Injury

Chymase Inhibition Attenuates Kidney Fibrosis in a Chronic Mouse Model of Renal Ischemia–Reperfusion Injury

IL-18 primes T cells with an antigen-inexperienced memory phenotype for proliferation and differentiation into effector cells through Notch signaling

Blastocyst-like Structures in the Peripheral Retina of Young Adult Beagles

Mechanisms of polyglycolic acid sheet-induced abdominal wall adhesions in hamsters.

Exploring the roles of SPARC as a proinflammatory factor and its potential as a novel therapeutic target against cardiovascular disease

<scp>TMEM182</scp> inhibits myocardial differentiation of human <scp>iPS</scp> cells by maintaining the activated state of Wnt/β‐catenin signaling through an increase in <scp>ILK</scp> expression

Collaborators (5)

Yoshimasa Tanaka

Hideki Tomiyama

Hidehiro Oku

Shinji Takai

TETSUO NAKATA

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