SHINSUKE NIWA

Associate Prof

Tohoku University

Japan

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Shinsuke Niwa is an Associate Professor at Tohoku University in Japan. His research primarily focuses on kinesin motor proteins, mitochondrial dynamics, and the intricacies of microtubule and actin interactions. Recent publications include studies on DNA origami for observing kinesin cooperation and the role of SLC-25A46 in neuronal morphology. His work contributes valuable insights into cellular transport mechanisms and their implications in neurobiology.

Articles (19)

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2025

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JAPAN

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Takako Kikkawa

Assistant Professor (Prominent Research Fellow)

Tohoku University graduate school of medicine

JAPAN

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Kozo Tanaka

JAPAN

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SHINSUKE NIWA

Research Interests

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About

Articles (19)

Dimerization of GAS2 mediates crosslinking of microtubules and F-actin

SLC-25A46 regulates mitochondrial fusion through the mitofusin protein FZO-1 and is essential for maintaining neuronal morphology.

Protocol for assembling photostable fluorescence-labeled tiny DNA origami blocks to observe kinesin cooperation

Modular photostable fluorescent DNA blocks dissect the effects of pathogenic mutant kinesin on collective transport

Biased movement of monomeric kinesin-3 KLP-6 explained by a symmetric Brownian ratchet model

Characterizing human KIF1Bß motor activity by single-molecule motility assays and Caenorhabtidis elegans genetics

The FHA domain is essential for the autoinhibition of KIF1A/UNC-104

Autoinhibition and activation of kinesin-1 and their involvement in amyotrophic lateral sclerosis

Comparative analysis of two Caenorhabditis elegans kinesins KLP-6 and UNC-104 reveals a common and distinct activation mechanism in kinesin-3

Comparative analysis of two Caenorhabditis elegans kinesins KLP-6 and UNC-104 reveals a common and distinct activation mechanism in kinesin-3

Kinesin-like motor protein KIF23 maintains neural stem and progenitor cell pools in the developing cortex

Generation of recombinant and chickenized scFv versions of an anti-kinesin monoclonal antibody H2.

Insight into the regulation of axonal transport from the study of KIF1A-associated neurological disorder

Dynein intermediate chains DYCI‐1 and WDR‐60 have specific functions in Caenorhabditis elegans

Modeling the motion of disease-associated KIF1A heterodimers

Synergistic autoinhibition and activation mechanisms control kinesin-1 motor activity

De novo mutations in KIF1A-associated neuronal disorder (KAND) dominant-negatively inhibit motor activity and axonal transport of synaptic vesicle precursors

An ALS‐associated KIF5A mutant forms oligomers and aggregates and induces neuronal toxicity

Neural and behavioral control in Caenorhabditis elegans by a yellow-light–activatable caged compound

Collaborators (9)

Minoru Kawatani

Masahide Kikkawa

Akihiro Narita

Atsushi Nakagawa

Toshiyuki Oda

Kyoko Chiba

Tsuyoshi Imasaki

Takako Kikkawa

Kozo Tanaka

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