Sho Hasegawa

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Assistant professor

University of Tokyo

Japan

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About

Dr. Sho Hasegawa is an Assistant Professor at The University of Tokyo, Japan. His research encompasses a range of areas including renal physiology, tissue imaging techniques, and the molecular mechanisms underlying kidney function and disease. His recent publications focus on the role of cellular mechanisms in kidney injury, the impact of systemic factors on kidney health, and innovative methods for assessing tissue conditions and responses.

Recent Grants

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Whole-kidney three-dimensional analysis for clarifying the pathophysiology of kidney diseases

Open Date: 2019-04-01

Close Date: 2021-03-01

Articles (15)

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2024

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2022

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2021

Collaborators (5)

Reiko Inagi

University of Tokyo

JAPAN

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Yoichi Takakusagi

National Institutes for Quantum and Radiological Science and Technology

JAPAN

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Minoru Kobayashi

特定助教 / Program-Specific Assistant Professor

Kyoto University

JAPAN

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Tsuyoshi Inoue

Professor

Nagasaki University

JAPAN

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Etsuo Susaki

Professor

Juntendo University

JAPAN

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Sho Hasegawa

Research Interests

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About

Recent Grants

Articles (15)

Whole-Body and Whole-Organ 3D Imaging of Hypoxia Using an Activatable Covalent Fluorescent Probe Compatible with Tissue Clearing.

Molecular mechanisms of kidney crosstalk with distant organs

Good and evil, more reasons to study UPR

Anti-aging mechanism of calorie restriction in humans

Lysophosphatidylcholine mediates fast decline in kidney function in diabetic kidney disease

Association Between Diabetes and Major Bleeding Complications of Renal Biopsy

Activation of α7 nicotinic acetylcholine receptors attenuates monocyte–endothelial adhesion through FUT7 inhibition

SGLT2 inhibition in chronic kidney disease: a preventive strategy against acute kidney injury at the same time?

The subtle long-lasting burden of mitochondrial DNA variants

Urinary single-cell transcriptomics: a promising noninvasive method for assessing acute kidney injury

Bone marrow stromal cell antigen-1 (CD157) regulated by sphingosine kinase 2 mediates kidney fibrosis

Whole-Kidney Three-Dimensional Staining with CUBIC

Decreased IFT88 expression with primary cilia shortening causes mitochondrial dysfunction in cisplatin-induced tubular injury

Activation of Sympathetic Signaling in Macrophages Blocks Systemic Inflammation and Protects against Renal Ischemia-Reperfusion Injury

Harnessing Metabolomics to Describe the Pathophysiology Underlying Progression in Diabetic Kidney Disease

Collaborators (5)

Reiko Inagi

Yoichi Takakusagi

Minoru Kobayashi

Tsuyoshi Inoue

Etsuo Susaki

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