Shu Kondo

Associate Professor

University of Tokyo

Japan

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Shu Kondo is an Associate Professor at Tokyo University of Science, Japan. His research primarily focuses on the molecular mechanisms in Drosophila, exploring areas such as cell fate determination, neuropeptide signaling, and apoptosis. Recent publications include studies on microtubule dynamics during dendrite pruning and the regulation of lipid metabolism in response to sugar. He is particularly interested in the Hippo pathway and its effects on organ size and cell identity.

Articles (10)

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2021

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2021

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2021

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2021

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2021

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2021

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2021

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2021

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2021

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2020

Collaborators (12)

Yuto Yoshinari

Assistant Professor

Gunma University

JAPAN

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Akira Nakamura

Professor

Kumamoto University

JAPAN

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Takumi Kamiyama

assistant professor

University of Tsukuba

JAPAN

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Fumiaki Obata

Visiting Associate Professor

Kyoto University

JAPAN

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Bruno Lemaitre

Professor

SWITZERLAND

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Ryusuke Niwa

Professor

University of Tsukuba

JAPAN

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Young-Joon Kim

Professor

Gwangju Institute of Science and Technology

SOUTH KOREA

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Michael Pankratz

Professor

University of Bonn

GERMANY

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Giovanni Bosco

Professor

Dartmouth College Geisel School of Medicine

UNITED STATES

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Hiromu Tanimoto

Tohoku University

JAPAN

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Yuko Shimada-Niwa

Assistant Professor

University of Tsukuba

JAPAN

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Florent Masson

University of Bristol

UNITED KINGDOM

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Shu Kondo

Research Interests

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About

Articles (10)

The sugar-responsive enteroendocrine neuropeptide F regulates lipid metabolism through glucagon-like and insulin-like hormones in Drosophila melanogaster

Neuropeptide F signaling regulates parasitoid-specific germline development and egg-laying in Drosophila

A secreted factor NimrodB4 promotes the elimination of apoptotic corpses by phagocytes in Drosophila

A systematic analysis of microtubule-destabilizing factors during dendrite pruning in Drosophila.

The Hippo pathway uses different machinery to control cell fate and organ size.

Crumbs and the apical spectrin cytoskeleton regulate R8 cell fate in the Drosophila eye.

A population of neurons that produce hugin and express the diuretic hormone 44 receptor gene projects to the corpora allata in Drosophila melanogaster.

Voluntary intake of psychoactive substances is regulated by the dopamine receptor Dop1R1 in Drosophila.

The neuropeptide allatostatin C from clock-associated DN1p neurons generates the circadian rhythm for oogenesis.

Sequential activation of transcriptional repressors promotes progenitor commitment by silencing stem cell identity genes.

Collaborators (12)

Yuto Yoshinari

Akira Nakamura

Takumi Kamiyama

Fumiaki Obata

Bruno Lemaitre

Ryusuke Niwa

Young-Joon Kim

Michael Pankratz

Giovanni Bosco

Hiromu Tanimoto

Yuko Shimada-Niwa

Florent Masson

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