Srinivas Mummidi

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Professor

Texas A&M University - San Antonio

United States

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Dr. Srinivas Mummidi is a Professor at Texas A&M University – San Antonio in the United States. His research areas encompass neurogenesis, cardiometabolic traits in diverse populations, obesity-related cardiovascular issues, and insulin sensitivity in pediatric populations. Dr. Mummidi's recent publications explore the interactions between diabetes, HIV, and obesity, as well as developmental programming effects of lactation.

Recent Grants

Grant: Close

Impact of disease associated noncoding polymorphisms on post-transcriptional gene regulation

Open Date: 2016-04-01

Close Date: 2020-03-31

Grant: Close

Transcriptional Mechanisms Regulating the HIV-1 coreceptor CCR5

Open Date: 2011-04-01

Close Date: 2015-03-31

Grant: Close

CCR5 Regulation and Promoter Variants in HIV-1

Open Date: 1998-07-01

Close Date: 2017-08-31

Articles (10)

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2023

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2022

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2021

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2021

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2021

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2021

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2021

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2021

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2021

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2020

Collaborators (8)

Takashi Ito

Professor

University of Kansas

UNITED STATES

Assistant Professor

The University of Texas Rio Grande Valley - Edinburg Campus

UNITED STATES

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Srinivas Mummidi

Research Interests

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About

Recent Grants

Articles (10)

Exosomes induce neurogenesis of pluripotent P19 cells.

Burden of Type 2 Diabetes and Associated Cardiometabolic Traits and Their Heritability Estimates in Endogamous Ethnic Groups of India: Findings From the INDIGENIUS Consortium.

Comparison of Carotid Intima-Media Thickness in Children and Adults With and Without Obesity: A Hysteresis Model.

Association of HIV-1 Infection and Antiretroviral Therapy With Type 2 Diabetes in the Hispanic Population of the Rio Grande Valley, Texas, USA.

Sacubitril/valsartan inhibits obesity-associated diastolic dysfunction through suppression of ventricular-vascular stiffness.

Further evidence supporting a potential role for ADH1B in obesity.

Serum carotenoids and Pediatric Metabolic Index predict insulin sensitivity in Mexican American children.

Lactation Plays a Fundamental Role in Developmental Programming.

The SGLT2 inhibitor Empagliflozin attenuates interleukin-17A-induced human aortic smooth muscle cell proliferation and migration by targeting TRAF3IP2/ROS/NLRP3/Caspase-1-dependent IL-1β and IL-18 secretion

Acanthosis nigricans as a composite marker of cardiometabolic risk and its complex association with obesity and insulin resistance in Mexican American children

Collaborators (8)

Takashi Ito

Dharambir K. Sanghera

Jose Canas

devjit tripathy

Joanne Curran

Satish Kumar

Swapan Das

Juan Lopez-Alvarenga

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