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Stefan Lechner

Professor

University Medical Center Hamburg-Eppendorf

Germany

Research Interests

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About

Stefan Lechner is a Professor at the University Medical Center Hamburg-Eppendorf in Germany. His research focuses on the mechanisms of pain and sensory neuron function, particularly related to mechanically activated ion channels and their implications in neuropathic pain. Recent articles highlight his contributions to understanding the role of various proteins and ion channels in sensory processing and blood pressure regulation.

Articles (12)

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2026

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2026

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2025

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2025

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2023

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2023

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2022

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2022

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2021

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2020

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2020

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2020

Collaborators (2)

Gary Lewin

Group Leader

Max Delbrück Center for Molecular Medicine in the Helmholtz Association

GERMANY

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Hongwei Zheng

Heidelberg University

GERMANY

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Stefan Lechner

Research Interests

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Articles (12)

State-dependent binding of the wedge domain controls inactivation of the mechanosensitive ion channel PIEZO1.

Oligodendrocyte mechanotransduction channel TMEM63A regulates myelin sheath geometry.

3D-MINFLUX nanoscopy reveals distinct allosteric mechanisms for activation and modulation of PIEZO1 by Yoda1.

Cluster nanoarchitecture and structural diversity of PIEZO1 at rest and during activation in intact cells

Role of TMEM100 in mechanically insensitive nociceptor un-silencing

Advances and recent insights into the gating mechanisms of the mechanically activated ion channels PIEZO1 and PIEZO2

Neuropathic pain caused by miswiring and abnormal end organ targeting.

Intrinsically disordered intracellular domains control key features of the mechanically-gated ion channel PIEZO2.

Identification of a population of peripheral sensory neurons that regulates blood pressure.

USH2A is a Meissner's corpuscle protein necessary for normal vibration sensing in mice and humans.

SUMOylation of Enzymes and Ion Channels in Sensory Neurons Protects against Metabolic Dysfunction, Neuropathy, and Sensory Loss in Diabetes.

TTX-Resistant Sodium Channels Functionally Separate Silent From Polymodal C-nociceptors.

Collaborators (2)

Gary Lewin

Hongwei Zheng

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