Takashi Yamada

Associate Professor

Sapporo Medical University

Japan

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Dr. Takashi Yamada is an Associate Professor at Sapporo Medical University, Japan. His research focuses on skeletal muscle adaptations, mitochondrial function, and fatigue resistance, as evidenced by his recent publications on high-intensity training and muscle recovery mechanisms. He investigates the impact of contractile load and training protocols on muscle performance, particularly in the context of muscular dystrophies and related conditions.

Articles (17)

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2025

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2025

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2025

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2025

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2025

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2024

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2024

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2023

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2023

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2022

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2022

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2022

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2021

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2021

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2021

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2021

Collaborators (10)

Gianluigi Pironti

Ass. Professor (Senior Researcher)

Karolinska Institutet

SWEDEN

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Mattias Carlstrom

Karolinska Institutet

SWEDEN

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Hidetoshi Sakurai

Kyoto University

JAPAN

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Arthur Cheng

Assistant Professor

York University

CANADA

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Stefano Gastaldello

Karolinska Institutet

SWEDEN

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Hakan Westerblad

Karolinska Institutet

SWEDEN

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Johanna Lanner

Karolinska Institutet

SWEDEN

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Noritsugu Tohse

Sapporo Medical University

JAPAN

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Lars H Lund

Karolinska University Hospital

SWEDEN

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Daniel Andersson

Karolinska University Hospital

SWEDEN

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Takashi Yamada

Research Interests

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About

Articles (17)

The contralateral repeated bout effect is not caused by adaptations in skeletal muscle

Fatigue Resistance and Mitochondrial Adaptations to Isometric Interval Training in Dystrophin‐Deficient Muscle: Role of Contractile Load

Polyplex Nanomicelle‐Mediated Pgc‐1α4 mRNA Delivery Via Hydrodynamic Limb Vein Injection Enhances Damage Resistance in Duchenne Muscular Dystrophy Mice

Effects of contraction frequency during high-intensity training on fatigue resistance and aerobic adaptations in mouse skeletal muscle

Task-Dependent Mechanisms Underlying Prolonged Low-Frequency Force Depression

High-intensity interval training using electrical stimulation ameliorates muscle fatigue in chronic kidney disease-related cachexia by restoring mitochondrial respiratory dysfunction

Platelet-rich plasma does not accelerate the healing of damaged muscle following muscle strain

Intrinsic contractile dysfunction due to impaired sarcoplasmic reticulum Ca2+ release in compensatory hypertrophied muscle fibers following synergist ablation

Skeletal muscle endurance declines with impaired mitochondrial respiration and inadequate supply of acetyl-CoA during muscle fatigue in 5/6 nephrectomized rats

High‐intensity interval training in the form of isometric contraction improves fatigue resistance in dystrophin‐deficient muscle

Citrullination is linked to reduced Ca2+ sensitivity in hearts of a murine model of rheumatoid arthritis

Dissociation of SH3 and cysteine-rich domain 3 and junctophilin 1 from dihydropyridine receptor in dystrophin-deficient muscles

Improved skeletal muscle fatigue resistance in experimental autoimmune myositis mice following high-intensity interval training.

Larger improvements in fatigue resistance and mitochondrial function with high‐ than with low‐intensity contractions during interval training of mouse skeletal muscle

Preconditioning contractions prevent prolonged force depression and Ca2+-dependent proteolysis of STAC3 after damaging eccentric contractions

Larger improvements in fatigue resistance and mitochondrial function with high‐ than with low‐intensity contractions during interval training of mouse skeletal muscle

Long-term wheel-running prevents reduction of grip strength in type 2 diabetic rats.

Collaborators (10)

Gianluigi Pironti

Mattias Carlstrom

Hidetoshi Sakurai

Arthur Cheng

Stefano Gastaldello

Hakan Westerblad

Johanna Lanner

Noritsugu Tohse

Lars H Lund

Daniel Andersson

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