Takumi Maruhashi

Assistant Professor

University of Tokyo

Japan

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Dr. Takumi Maruhashi is an Assistant Professor at the University of Tokyo in Japan. His research focuses on immune regulation, specifically exploring the roles of various receptors and signaling pathways in T cell function, autoimmunity, and osteoclastogenesis. Recent publications highlight his work on DCIR, LAG-3, and PD-1, contributing significantly to the understanding of immune responses in both autoimmunity and cancer.

Articles (10)

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2023

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2022

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2022

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2022

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2021

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2021

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2021

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2021

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2020

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2020

Collaborators (11)

yoichiro iwakura

Professor

University of Tokyo

JAPAN

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Tatsuya Takemoto

Professor

Tokushima University

JAPAN

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Daisuke Sugiura

JAPAN

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Soo-Hyun Chung

Research Professor

University of Ulsan College of Medicine

SOUTH KOREA

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Harunori Yoshikawa

Assistant Professor

Tokushima University

JAPAN

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katsumi maenaka

Hokkaido University

JAPAN

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Kenji Shimizu

University of Tokyo

JAPAN

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Akimasa Seno

Okayama University Graduate School of Medicine, Dentistry and Pharmaceutical Sciences

JAPAN

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Shinobu Saijo

Associate Professor

Chiba University

JAPAN

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Noriyuki Fujikado

UNITED STATES

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Taku Okazaki

Professor

University of Tokyo

JAPAN

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Takumi Maruhashi

Research Interests

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Articles (10)

DCIR suppresses osteoclastic proliferation and resorption by downregulating M-CSF and RANKL signaling

Binding of LAG-3 to stable peptide-MHC class II limits T cell function and suppresses autoimmunity and anti-cancer immunity.

PD-1 agonism by anti-CD80 inhibits T cell activation and alleviates autoimmunity.

The C-type lectin receptor Clec1A plays an important role in the development of experimental autoimmune encephalomyelitis by enhancing antigen presenting ability of dendritic cells and inducing inflammatory cytokine IL-17.

DCIR and its ligand asialo-biantennary N-glycan regulate DC function and osteoclastogenesis.

T-cell-intrinsic and -extrinsic regulation of PD-1 function.

PD-1 preferentially inhibits the activation of low-affinity T cells.

TARM1 contributes to development of arthritis by activating dendritic cells through recognition of collagens.

LAG-3: from molecular functions to clinical applications.

PD-1 Imposes Qualitative Control of Cellular Transcriptomes in Response to T Cell Activation.

Collaborators (11)

yoichiro iwakura

Tatsuya Takemoto

Daisuke Sugiura

Soo-Hyun Chung

Harunori Yoshikawa

katsumi maenaka

Kenji Shimizu

Akimasa Seno

Shinobu Saijo

Noriyuki Fujikado

Taku Okazaki

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