Tara Doucet-O'Hare

Assistant Professor

Clemson University

United States

Research Interests

Explore related searches

Professors in United States Professors in Cancer Biology Cancer Biology professors in United States

Contact this professor

Get a free first email built on one of their papers, ready for you to edit and send yourself.

About

Dr. Tara Doucet-O'Hare is an Assistant Professor at Clemson University, USA. Her research areas include the role of endogenous retroviruses in cancer, particularly focusing on TDP-43 proteinopathy in amyotrophic lateral sclerosis and glioblastoma. She has contributed to advancements in understanding the relationship between human endogenous retrovirus K and cancer stemness, as well as strategies for drug repositioning in glioblastoma treatment.

Articles (10)

Year:

2024

Year:

2024

Year:

2023

Year:

2022

Year:

2022

Year:

2022

Year:

2021

Year:

2021

Year:

2021

Year:

2021

Collaborators (4)

Mynor Mendez Valdez

University of Miami Miller School of Medicine

UNITED STATES

View details

Vaidya Govindarajan

University of Miami

UNITED STATES

View details

Jared Rosenblum

UNITED STATES

View details

John Heiss

Head, Clinical Unit, and Neurological Surgery Residency Program Director

National Institutes of Health

UNITED STATES

View details

ApplyKite TwinLearns as you apply

Make your next move.

Prepare your application

Review drafts before you send them.

Tara Doucet-O'Hare

Research Interests

Explore related searches

Contact this professor

About

Articles (10)

TDP-43 proteinopathy in ALS is triggered by loss of ASRGL1 and associated with HML-2 expression

Antiretroviral Drug Repositioning for Glioblastoma

Human endogenous retrovirus K contributes to a stem cell niche in glioblastoma

The Role of HERV-K in Cancer Stemness.

Human Endogenous Retrovirus K Envelope in Spinal Fluid of Amyotrophic Lateral Sclerosis Is Toxic.

Differential expression of an endogenous retroviral element [HERV-K(HML-6)] is associated with reduced survival in glioblastoma patients.

Endogenous Retroviral Elements in Human Development and Central Nervous System Embryonal Tumors

SMARCB1 deletion in atypical teratoid rhabdoid tumors results in human endogenous retrovirus K (HML-2) expression.

A multiparametric pharmacogenomic strategy for drug repositioning predicts therapeutic efficacy for glioblastoma cell lines.

CSIG-24. TARGETED INHIBITION OF CDK5-MEDIATED REGULATION OF HUMAN ENDOGENOUS RETROVIRUS K (HML-2) ENVELOPE PROTEIN IN ATYPICAL TERATOID RHABDOID TUMOR

Collaborators (4)

Mynor Mendez Valdez

Vaidya Govindarajan

Jared Rosenblum

John Heiss

Make your next move.