Tenpei Akita

Professor

Hamamatsu Ika Daigaku

Japan

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Professor Tenpei Akita is a faculty member at Hamamatsu Ika Daigaku in Japan. His research focuses on neuronal excitability, ion channel functionality, and the effects of magnetic field stimulation on neuronal activity. Recent publications include studies on the role of WNK3 kinase, the impact of taurine depletion on neocortical neurons, and the significance of ATP6V0A1 in brain development.

Articles (10)

Year:

2024

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2024

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2023

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2023

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2022

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2022

Year:

2022

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2021

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2021

Year:

2020

Collaborators (3)

Hirotomo Saitsu

Hamamatsu University School of Medicine

JAPAN

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Mitsuhiro Kato

Professor

Showa University

JAPAN

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ADYA SARAN SINHA

Assistant Professor

Hamamatsu University School of Medicine

JAPAN

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Tenpei Akita

Research Interests

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About

Articles (10)

Static Magnetic Field Stimulation Enhances Shunting Inhibition via a SLC26 Family Cl<sup>-</sup> Channel, Inducing Intrinsic Plasticity.

Static Magnetic Field Stimulation Enhances Shunting Inhibition via a SLC26 Family Cl- Channel, Inducing Intrinsic Plasticity

A novel de novo KCNB1 variant altering channel characteristics in a patient with periventricular heterotopia, abnormal corpus callosum, and mild seizure outcome

A novel de novo KCNB1 variant altering channel characteristics in a patient with periventricular heterotopia, abnormal corpus callosum, and mild seizure outcome

WNK3 kinase maintains neuronal excitability by reducing inwardly rectifying K<sup>+</sup> conductance in layer V pyramidal neurons of mouse medial prefrontal cortex.

WNK3 kinase maintains neuronal excitability by reducing inwardly rectifying K+ conductance in layer V pyramidal neurons of mouse medial prefrontal cortex

Taurine depletion during fetal and postnatal development blunts firing responses of neocortical layer II/III pyramidal neurons

ATP6V0A1 encoding the a1-subunit of the V0 domain of vacuolar H+-ATPases is essential for brain development in humans and mice

ATP6V0A1 encoding the a1-subunit of the V0 domain of vacuolar H+-ATPases is essential for brain development in humans and mice

Intracellular Cl− dysregulation causing and caused by pathogenic neuronal activity

Collaborators (3)

Hirotomo Saitsu

Mitsuhiro Kato

ADYA SARAN SINHA

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