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TERESITA PADILLA-BENAVIDES

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Assistant Professor

Wesleyan University

United States

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Mechanisms of Cu-binding factors to promote myogenic gene expression

Open Date: 2021-08-01

Close Date: 2026-04-30

Articles (15)

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2026

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2021

Collaborators (11)

Marco Messina

Assistant Professor

University of Delaware

UNITED STATES

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Kyle Schachtschneider

Research Assistant Professor

University of Chicago

UNITED STATES

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isabelle hininger-favier

FRANCE

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Sylvie Coscoy

Centre National de la Recherche Scientifique (CNRS)

FRANCE

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Anthony N. Imbalzano

University of Massachusetts Chan Medical School

UNITED STATES

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Julie Gavard

CNRS

FRANCE

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Claire CALMEL

INSERM

FRANCE

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Donita C. Brady

University of Pennsylvania

UNITED STATES

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Uwe Schlattner

Université Grenoble Alpes

FRANCE

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Lobna Elkhadragy

Research Assistant Professor

The University of Illinois at Chicago

UNITED STATES

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Béatrice NAWROCKI-RABY

FRANCE

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TERESITA PADILLA-BENAVIDES

Research Interests

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Recent Grants

Articles (15)

Tceal7 is a BRG1-regulated target of calcineurin signaling that promotes myoblast differentiation

A periplasmic metallochaperone (PmcY) couples Zn 2+ transport to sensing in Pseudomonas aeruginosa

CuiT is a Cu importer required for metal homeostasis in Salmonella enterica

Cell Death Induced by Homoisoflavonoid Brazilin and Its Semisynthetic Derivatives on MDA-MB-231 and MCF7 Breast Cancer Cell Lines

A histochemical approach to activity-based copper sensing reveals cuproplasia-dependent vulnerabilities in cancer

Functional effect of indole-3 carbinol in the viability and invasive properties of cultured cancer cells

Differential Contributions of mSWI/SNF Chromatin Remodeler Sub-Families to Myoblast Differentiation

The Oncopig as an Emerging Model to Investigate Copper Regulation in Cancer

Differential requirements for different subfamilies of the mammalian SWI/SNF chromatin remodeling enzymes in myoblast cell cycle progression and expression of the Pax7 regulator

ZIP11 Regulates Nuclear Zinc Homeostasis in HeLa Cells and Is Required for Proliferation and Establishment of the Carcinogenic Phenotype

The molecular and cellular basis of copper dysregulation and its relationship with human pathologies

The mitochondrially-localized nucleoside diphosphate kinase D (NME4) is a novel metastasis suppressor

Loss of the Metastasis Suppressor NME1, But Not of Its Highly Related Isoform NME2, Induces a Hybrid Epithelial–Mesenchymal State in Cancer Cells

Editorial: Repurposed Drugs Targeting Cancer Signaling Pathways: Clinical Insights to Improve Oncologic Therapies

Editorial: Repurposed Drugs Targeting Cancer Signaling Pathways: Dissecting New Mechanism of Action Through In Vitro and In Vivo Analyses

Collaborators (11)

Marco Messina

Kyle Schachtschneider

isabelle hininger-favier

Sylvie Coscoy

Anthony N. Imbalzano

Julie Gavard

Claire CALMEL

Donita C. Brady

Uwe Schlattner

Lobna Elkhadragy

Béatrice NAWROCKI-RABY

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