Thomas Friedman

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Laboratory Chief

National Institutes of Health

United States

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About

Thomas Friedman is the Laboratory Chief at the National Institutes of Health Office of History in the United States. His research focuses on the mechanisms underlying hearing loss, including the molecular pathways involved in hair cell function and integrity. His recent publications explore topics such as myosin motor functions, genetic variants associated with deafness, and the structural properties of inner ear cells.

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Hereditary deafness

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Articles (10)

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2025

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2025

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2024

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2024

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2022

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2021

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2021

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2020

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2020

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2020

Collaborators (8)

Claudio Anselmi

Assistant Research Professor

George Washington University

UNITED STATES

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Naoki Watanabe

Professor

Kyoto University

JAPAN

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William Newman

The University of Manchester

UNITED KINGDOM

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Ivan Lopez

Adjunct Professor

David Geffen School of Medicine at UCLA

UNITED STATES

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Jonathan Bird

Assistant Professor

University of Florida

UNITED STATES

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S. Amer Riazuddin

Johns Hopkins University

UNITED STATES

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narinobu harada

Kindai University

JAPAN

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Gregory Frolenkov

Associate Professor

University of Kentucky

UNITED STATES

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Thomas Friedman

Research Interests

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About

Recent Grants

Articles (10)

Single-molecule fluorescence microscopy reveals regulatory mechanisms of MYO7A-driven cargo transport in stereocilia of live inner ear hair cells

MSRB3 antioxidant activity is necessary for inner ear cuticular plate structure and hair bundle integrity

Homozygous novel truncating variant of CLPP associated with severe Perrault syndrome

Deafness DFNB128 Associated with a Recessive Variant of Human MAP3K1 Recapitulates Hearing Loss of Map3k1-Deficient Mice

Unbalanced bidirectional radial stiffness gradients within the organ of Corti promoted by TRIOBP

The ATPase mechanism of myosin 15, the molecular motor mutated in DFNB3 human deafness

Fast-dissociating but highly specific antibodies are novel tools in biology, especially useful for multiplex super-resolution microscopy

Mouse Models of Human Pathogenic Variants of TBC1D24 Associated with Non-Syndromic Deafness DFNB86 and DFNA65 and Syndromes Involving Deafness

Atypical and ultra-rare Usher syndrome: a review

Growth factor and receptor malfunctions associated with human genetic deafness

Collaborators (8)

Claudio Anselmi

Naoki Watanabe

William Newman

Ivan Lopez

Jonathan Bird

S. Amer Riazuddin

narinobu harada

Gregory Frolenkov

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