Thomas Sanderson

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Associate Professor

University of Michigan Medical School

United States

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About

Thomas Sanderson is an Associate Professor at the University of Michigan Medical School in the United States. His research focuses on mitochondrial function, including oxidative phosphorylation regulation, the role of magnesium in cardiac arrest, and the interplay between oxidative stress and mitochondrial dynamics. He has published extensively on topics such as mass spectrometry processes, leukocyte modulation during cardiopulmonary resuscitation, and the implications of iron in T cell metabolism.

Recent Grants

Grant: Close

Non-invasive mitochondrial modulation therapy for ischemic stroke

Open Date: 2021-03-01

Close Date: 2026-02-28

Grant: Close

Early Neuroprotective Therapies for Out-of-Hospital Cardiac Arrest

Open Date: 2019-07-01

Close Date: 2023-06-30

Grant: Close

Infant NeuroLUX: A Novel Non-invasive Therapeutic Device for Neonatal Hypoxic Brain Injury

Open Date: 2018-04-01

Close Date: 2021-03-01

Grant: Close

Preventing Ischemia/Reperfusion Damage via Noninvasive Mitochondria-Targeted Therapy

Open Date: 2016-09-01

Close Date: 2019-08-01

Grant: Close

Limiting Brain Reperfusion Injury by Controlling Mitochondrial Function

Open Date: 2015-09-01

Close Date: 2019-06-01

Articles (21)

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2020

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2020

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2020

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2020

Collaborators (10)

Maik Hüttemann

Professor

Wayne State University School of Medicine

UNITED STATES

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Tatsuya Yamada

Assistant Professor

University of Nebraska - Lincoln

UNITED STATES

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Joseph Wider

Research Assistant Professor

University of Michigan

UNITED STATES

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Mohamad Tiba

University of Michigan

UNITED STATES

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Irene Díaz-Moreno

Full Professor

Universidad de Sevilla

SPAIN

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Garrett Fogo

University of Michigan

UNITED STATES

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Rachel Reinert

Assistant Professor

University of Michigan

UNITED STATES

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Ekta Shah

Assistant Teaching Professor

Northeastern University

CANADA

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Katlynn Emaus

University of Michigan

UNITED STATES

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Cindy Hsu

Assistant Professor

University of Michigan

UNITED STATES

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Thomas Sanderson

Research Interests

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About

Recent Grants

Articles (21)

Phosphorylations and Acetylations of Cytochrome <i>c</i> Control Mitochondrial Respiration, Mitochondrial Membrane Potential, Energy, ROS, and Apoptosis

Leukocyte filtration and leukocyte modulation therapy during extracorporeal cardiopulmonary resuscitation in a porcine model of prolonged cardiac arrest.

The role of magnesium in cardiac arrest.

ER-associated degradation adapter Sel1L is required for CD8<sup>+</sup> T cell function and memory formation following acute viral infection.

Iron regulates the quiescence of naive CD4 T cells by controlling mitochondria and cellular metabolism.

Mitochondrial membrane potential and oxidative stress interact to regulate Oma1‐dependent processing of Opa1 and mitochondrial dynamics

Regulation of mitochondrial oxidative phosphorylation through tight control of cytochrome c oxidase in health and disease - Implications for ischemia/reperfusion injury, inflammatory diseases, diabetes, and cancer.

New Processes for Ionizing Nonvolatile Compounds in Mass Spectrometry: The Road of Discovery to Current State-of-the-Art.

Cytochrome c lysine acetylation regulates cellular respiration and cell death in ischemic skeletal muscle

Modulation of mitochondrial function with near-infrared light reduces brain injury in a translational model of cardiac arrest.

Non-invasive treatment of ischemia/reperfusion injury: Effective transmission of therapeutic near-infrared light into the human brain through soft skin-conforming silicone waveguides.

Sometimes less is more: inhibitory infrared light during early reperfusion calms hyperactive mitochondria and suppresses reperfusion injury.

Does Disruption of Optic Atrophy-1 (OPA1) Contribute to Cell Death in HL-1 Cardiomyocytes Subjected to Lethal Ischemia-Reperfusion Injury?

Rapid Treatment with Intramuscular Magnesium Sulfate During Cardiopulmonary Resuscitation Does Not Provide Neuroprotection Following Cardiac Arrest.

Inhibiting Mitochondrial Cytochrome <i>c</i> Oxidase Downregulates Gene Transcription After Traumatic Brain Injury in <i>Drosophila</i>.

Machine learning-based classification of mitochondrial morphology in primary neurons and brain.

Mitochondrial fission and mitophagy are independent mechanisms regulating ischemia/reperfusion injury in primary neurons

Endoplasmic reticulum-associated degradation regulates mitochondrial dynamics in brown adipocytes.

Aerosol generation during chest compression and defibrillation in a swine cardiac arrest model.

Cytochrome c oxidase-modulatory near-infrared light penetration into the human brain: Implications for the noninvasive treatment of ischemia/reperfusion injury.

Non-invasive treatment with near-infrared light: A novel mechanisms-based strategy that evokes sustained reduction in brain injury after stroke

Collaborators (10)

Maik Hüttemann

Tatsuya Yamada

Joseph Wider

Mohamad Tiba

Irene Díaz-Moreno

Garrett Fogo

Rachel Reinert

Ekta Shah

Katlynn Emaus

Cindy Hsu

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