Thomas Wieland

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Professor and Chair

Ruprecht-Karls-Universität Heidelberg Medizinische Fakultät Mannheim

Germany

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About

Professor Thomas Wieland serves as the Chair at Ruprecht-Karls-Universität Heidelberg Medizinische Fakultät Mannheim in Germany. His research focuses on vascular biology, cancer metabolism, and neurovascular unit maintenance, as demonstrated in recent studies on transcriptional regulation and signaling pathways. His work includes exploring mechanisms underlying vessel growth and the role of nucleoside diphosphate kinases in cancer metastasis.

Recent Grants

Grant: Close

Interaction/Oligomerization of Nucleoside Diphosphate Kinase Isoforms in Pathological Processes

Open Date: 2022-10-01

Close Date: 2025-09-01

Grant: Close

GRK 2816: Non-canonical G protein signaling pathways: Mechanisms, functions, consequences

Open Date: 2022-01-01

Close Date:

Grant: Close

Ambulante Integrierte Gesundheitszentren zur Optimierung der ärztlichen Versorgung und Pflege im ländlichen Raum (AMBIGOAL)

Open Date: 2019-04-01

Close Date: 2021-03-01

Grant: Close

Regulation of the tumor microenvironment by chronic β‑adrenergic stress in the tumor vasculature

Open Date: 2019-01-01

Close Date: 2022-12-01

Grant: Close

GRK 1874: Diabetic Microvascular Complications (DIAMICOM)

Open Date: 2013-01-01

Close Date:

Articles (16)

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2024

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2024

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2022

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2022

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2022

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2022

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2022

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2022

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2022

Collaborators (6)

Felix A. Trogisch

Ruprecht-Karls-Universität Heidelberg Medizinische Fakultät Mannheim

GERMANY

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Raquel Mejías-Luque

Ludwig-Maximilians-Universität München

GERMANY

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Sebastiaan Werten

Medical University of Innsbruck

AUSTRIA

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Richmond Muimo

UNITED KINGDOM

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Clara Baldin

University of Innsbruck

AUSTRIA

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Magdolna Levay

Universitätsmedizin Mannheim

GERMANY

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Thomas Wieland

Research Interests

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About

Recent Grants

Articles (16)

Mechanistic Insights into Substrate Recognition of Human Nucleoside Diphosphate Kinase C Based on Nucleotide-Induced Structural Changes

RNF20-mediated transcriptional pausing and VEGFA splicing orchestrate vessel growth

PRUNE1 and NME/NDPK family proteins influence energy metabolism and signaling in cancer metastases

Pathogen and human NDPK-proteins promote AML cell survival via monocyte NLRP3-inflammasome activation

Contribution of the hexosamine biosynthetic pathway in the hyperglycemia-dependent and -independent breakdown of the retinal neurovascular unit

Chemical Tools for study of Phosphohistidine: Generation of Selective Τ‐phosphohistidine and Π‐phosphohistidine Antibodies

Sufficient Cav-1 levels in the endothelium are critical for the maintenance of the neurovascular unit in the retina

The Muscarinic Acetylcholine M2 Receptor-Induced Nitration of p190A by eNOS Increases RhoA Activity in Cardiac Myocytes

Tankyrase inhibition interferes with junction remodeling, induces leakiness, and disturbs YAP1/TAZ signaling in the endothelium

Chronic isoprenaline/phenylephrine vs. exclusive isoprenaline stimulation in mice: critical contribution of alpha1-adrenoceptors to early cardiac stress responses

RGS3L allows for an M2 muscarinic receptor-mediated RhoA-dependent inotropy in cardiomyocytes

Lamin A/C-dependent chromatin architecture safeguards naïve pluripotency to prevent aberrant cardiovascular cell fate and function

Functional antagonism between CagA and DLC1 in gastric cancer

Functional antagonism between CagA and DLC1 in gastric cancer

The orphan receptor GPRC5B activates pro-inflammatory signaling in the vascular wall via Fyn and NFκB

The orphan receptor GPRC5B activates pro-inflammatory signaling in the vascular wall via Fyn and NFkB

Collaborators (6)

Felix A. Trogisch

Raquel Mejías-Luque

Sebastiaan Werten

Richmond Muimo

Clara Baldin

Magdolna Levay

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