Timothy Crother

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Adjunct Professor

University of California, Los Angeles

United States

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Timothy Crother is an Adjunct Professor at the University of California Los Angeles, USA. His research focuses on immunopathology, cardiovascular inflammation, and the role of the intestinal microbiota in various diseases, as highlighted in his recent articles. He has also explored the impacts of hypothermia on lung injury and the mechanisms of interleukin signaling in conditions such as Kawasaki Disease.

Recent Grants

Grant: Close

The role of Chlamydia pneumoniae infection in Alzheimer's Disease

Open Date: 2021-10-01

Close Date: 2026-06-01

Grant: Close

Chlamydia pneumoniae infection accelerated Alzheimer’s Disease.

Open Date: 2021-10-01

Close Date: 2024-09-01

Grant: Close

Role of Rip2 in the Generation of Pathogenic Th17/Th1 T-cells and Ileitis

Open Date: 2020-12-07

Close Date: 2022-11-30

Articles (22)

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2020

Collaborators (6)

Elsa Sanchez-Lopez

Assistant Professor

University of California, San Diego

UNITED STATES

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Alexandra Rundberg Nilsson

Lund University

SWEDEN

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Magali Noval Rivas

Assistant Professor

Cedars-Sinai Medical Center

UNITED STATES

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Stuart Lipton

University of California San Diego School of Medicine

UNITED STATES

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David Zemmour

University of Chicago

UNITED STATES

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Caroline Jefferies

Associate Professor

Cedars-Sinai Medical Center

UNITED STATES

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Timothy Crother

Research Interests

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Recent Grants

Articles (22)

pDCs drive immunopathology by sensing oxidized mitochondrial DNA

Hypothermia protects against ventilator-induced lung injury by limiting IL-1β release and NETs formation

Hypothermia protects against ventilator-induced lung injury by limiting IL-1β release and NETs formation

Interleukin‐1 Signaling on Vascular Smooth Muscle Cells Accelerates Atherosclerosis in a Murine Model of Kawasaki Disease

Intestinal Microbiota Contributes to the Development of Cardiovascular Inflammation and Vasculitis in Mice.

Long-term cardiovascular inflammation and fibrosis in a murine model of vasculitis induced by Lactobacillus casei cell wall extract

<i>Chlamydia pneumoniae</i> in Alzheimer's disease pathology.

The intestinal microbiota contributes to the development of immune-mediated cardiovascular inflammation and vasculitis in mice

Mitochondrial quality control in health and cardiovascular diseases.

IL-33 Promotes Cardiovascular Lesions Development In A Mouse Model Of Kawasaki Disease

IL-33 promotes cardiovascular lesions development in a mouse model of Kawasaki Disease

Il-1 Signaling On Vascular Smooth Muscle Cells Accelerates Atherosclerosis After Murine Kawasaki Disease Vasculitis

Metformin inhibition of mitochondrial ATP and DNA synthesis abrogates NLRP3 inflammasome activation and pulmonary inflammation

NLRP3 Inflammasome Mediates Immune-Stromal Interactions in Vasculitis.

NLRP3 Inflammasome Mediates Immune-Stromal Interactions in Vasculitis

Autophagy-mitophagy induction attenuates cardiovascular inflammation in a murine model of Kawasaki disease vasculitis.

Recruitment of pro-IL-1α to mitochondrial cardiolipin, via shared LC3 binding domain, inhibits mitophagy and drives maximal NLRP3 activation

Oxidative DNA Damage Accelerates Skin Inflammation in Pristane-Induced Lupus Model

MD-2 as a possible therapeutic target for atherosclerosis

Sex-Specific Effects of the Nlrp3 Inflammasome on Atherogenesis in LDL Receptor-Deficient Mice

Loss of testosterone impairs anti-tumor neutrophil function

Autophagy Protects Against Developing Increased Lung Permeability and Hypoxemia by Down Regulating Inflammasome Activity and IL-1β in LPS Plus Mechanical Ventilation-Induced Acute Lung Injury

Collaborators (6)

Elsa Sanchez-Lopez

Alexandra Rundberg Nilsson

Magali Noval Rivas

Stuart Lipton

David Zemmour

Caroline Jefferies

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