Tomoya Fujie

Junior Associate Professor

University of Tokyo

Japan

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About

Dr. Tomoya Fujie is a Junior Associate Professor at Tokyo University of Science, Japan. His research focuses on the cellular and molecular mechanisms of endothelial cells, particularly relating to the effects of toxic substances such as methylmercury and cadmium. He has published several articles investigating purinergic receptors, VEGF-A expression, and the impact of heavy metals on vascular endothelial cell functionality.

Articles (19)

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2025

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2020

Collaborators (4)

Yasuyuki Fujiwara

Professor

Tokyo University of Pharmacy and Life Sciences

JAPAN

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Yoshito Kumagai

Professor

University of Tsukuba

JAPAN

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Yo Shinoda

Associate Professor

Tokyo University of Pharmacy and Life Sciences

JAPAN

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Toshiyuki Kaji

University of Tokyo

JAPAN

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Tomoya Fujie

Research Interests

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About

Articles (19)

Expression profiles of purinergic P1 and P2 receptors in cultured bovine aortic endothelial cells, bovine aortic smooth muscle cells, and human vascular endothelial EA.hy926 cells

Induction of VEGF-A expression by methylmercury is mediated by the EGFR–p38 MAPK–COX-2–PKA pathway in cultured human brain microvascular pericytes

Mechanisms Underlying Sensory Nerve-Predominant Damage by Methylmercury in the Peripheral Nervous System

Cadmium Induces Vascular Endothelial Cell Detachment by Downregulating Claudin-5 and ZO-1 Levels

Comparative study of susceptibility to methylmercury cytotoxicity in cell types composing rat peripheral nerves: a higher susceptibility of dorsal root ganglion neurons

Transcriptome analysis of cultured human vascular endothelial cells after γ-ray irradiation and correlation analysis with ATP, ADP, and adenosine as secondary soluble factors

Protection of cultured vascular endothelial cells against cadmium cytotoxicity by simultaneous treatment or pretreatment with manganese

Zrt- and Irt-like protein 8 (ZIP8) and metallothionein aid in zinc protection of cultured vascular endothelial cells against cadmium cytotoxicity

Pathogenesis of selective damage of granule cell layer in cerebellum of rats exposed to methylmercury

Lead suppresses perlecan expression via EGFR-ERK1/2-COX-2-PGI<sub>2</sub> pathway in cultured bovine vascular endothelial cells

Lead induces a metal transporter, ZIP8, via activation of the NF-κB signaling pathway and the induction is involved in the protection against lead cytotoxicity by intracellular lead accumulation independent mechanisms in cultured vascular endothelial cells

Induction of ZIP8, a ZIP transporter, via NF-κB signaling by the activation of IκBα and JNK signaling in cultured vascular endothelial cells exposed to cadmium

TGF-β1 Potentiates the Cytotoxicity of Cadmium by Induction of a Metal Transporter, ZIP8, Mediated by the ALK5-Smad2/3 and ALK5-Smad3-p38 MAPK Signal Pathways in Cultured Vascular Endothelial Cells

Fibroblast Growth Factor-2 Upregulates Reactive Sulfur Species Production <i>via</i> ERK1/2 Signal-Mediated Cystathionine γ-Lyase Induction in Cultured Bovine Aortic Endothelial Cells

Synthesis of Reactive Sulfur Species in Cultured Vascular Endothelial Cells after Exposure to TGF-β1: Induction of Cystathionine γ-Lyase and Cystathionine β-Synthase Expression Mediated by the ALK5-Smad2/3/4 and ALK5-Smad2/3-ATF4 Pathways

Sodium trisulfide, a sulfane sulfur donor, stimulates bovine aortic endothelial cell proliferation in culture

Cadmium induces plasminogen activator inhibitor-1 via Smad2/3 signaling pathway in human endothelial EA.hy926 cells

Induction of metallothionein isoforms in cultured bovine aortic endothelial cells exposed to cadmium

Zn(ii)2,9-dimethyl-1,10-phenanthroline stimulates cultured bovine aortic endothelial cell proliferation

Collaborators (4)

Yasuyuki Fujiwara

Yoshito Kumagai

Yo Shinoda

Toshiyuki Kaji

Make your next move.

Tomoya Fujie

Research Interests

Explore related searches

Contact this professor

About

Articles (19)

Expression profiles of purinergic P1 and P2 receptors in cultured bovine aortic endothelial cells, bovine aortic smooth muscle cells, and human vascular endothelial EA.hy926 cells

Induction of VEGF-A expression by methylmercury is mediated by the EGFR–p38 MAPK–COX-2–PKA pathway in cultured human brain microvascular pericytes

Mechanisms Underlying Sensory Nerve-Predominant Damage by Methylmercury in the Peripheral Nervous System

Cadmium Induces Vascular Endothelial Cell Detachment by Downregulating Claudin-5 and ZO-1 Levels

Comparative study of susceptibility to methylmercury cytotoxicity in cell types composing rat peripheral nerves: a higher susceptibility of dorsal root ganglion neurons

Transcriptome analysis of cultured human vascular endothelial cells after γ-ray irradiation and correlation analysis with ATP, ADP, and adenosine as secondary soluble factors

Protection of cultured vascular endothelial cells against cadmium cytotoxicity by simultaneous treatment or pretreatment with manganese

Zrt- and Irt-like protein 8 (ZIP8) and metallothionein aid in zinc protection of cultured vascular endothelial cells against cadmium cytotoxicity

Pathogenesis of selective damage of granule cell layer in cerebellum of rats exposed to methylmercury

Lead suppresses perlecan expression via EGFR-ERK1/2-COX-2-PGI&lt;sub&gt;2&lt;/sub&gt; pathway in cultured bovine vascular endothelial cells

Lead induces a metal transporter, ZIP8, via activation of the NF-κB signaling pathway and the induction is involved in the protection against lead cytotoxicity by intracellular lead accumulation independent mechanisms in cultured vascular endothelial cells

Induction of ZIP8, a ZIP transporter, via NF-κB signaling by the activation of IκBα and JNK signaling in cultured vascular endothelial cells exposed to cadmium

TGF-β1 Potentiates the Cytotoxicity of Cadmium by Induction of a Metal Transporter, ZIP8, Mediated by the ALK5-Smad2/3 and ALK5-Smad3-p38 MAPK Signal Pathways in Cultured Vascular Endothelial Cells

Fibroblast Growth Factor-2 Upregulates Reactive Sulfur Species Production <i>via</i> ERK1/2 Signal-Mediated Cystathionine γ-Lyase Induction in Cultured Bovine Aortic Endothelial Cells

Synthesis of Reactive Sulfur Species in Cultured Vascular Endothelial Cells after Exposure to TGF-β1: Induction of Cystathionine γ-Lyase and Cystathionine β-Synthase Expression Mediated by the ALK5-Smad2/3/4 and ALK5-Smad2/3-ATF4 Pathways

Sodium trisulfide, a sulfane sulfur donor, stimulates bovine aortic endothelial cell proliferation in culture

Cadmium induces plasminogen activator inhibitor-1 via Smad2/3 signaling pathway in human endothelial EA.hy926 cells

Induction of metallothionein isoforms in cultured bovine aortic endothelial cells exposed to cadmium

Zn(ii)2,9-dimethyl-1,10-phenanthroline stimulates cultured bovine aortic endothelial cell proliferation

Collaborators (4)

Yasuyuki Fujiwara

Yoshito Kumagai

Yo Shinoda

Toshiyuki Kaji

Make your next move.

Lead suppresses perlecan expression via EGFR-ERK1/2-COX-2-PGI<sub>2</sub> pathway in cultured bovine vascular endothelial cells