Toshiyuki Itai

Assistant Professor

Yokohama National University

Japan

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Toshiyuki Itai is an Assistant Professor at Yokohama City University in Japan. His research focuses on the impacts of genetic mutations on brain development and congenital malformations, as well as the effects of body composition on chemotherapy toxicity. His recent publications explore topics such as gene functions in orofacial clefts, single-cell expression profiles, and neurodevelopmental disorders.

Articles (13)

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2025

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2023

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2023

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2023

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2022

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2021

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2021

Collaborators (7)

Yoshihiro Watanabe

Yokohama City University Medical Center

JAPAN

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Atsushi Takata

JAPAN

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Lukas Simon

Research Assistant Professor

The University of Texas Health Science Center at Houston

UNITED STATES

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yulin dai

The University of Texas Health Science Center at Houston

UNITED STATES

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Yohei Nitta

Niigata University

JAPAN

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Jingchun Chen

assistant professor

University of Nevada

UNITED STATES

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Mitsuyoshi Suzuki

JAPAN

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Toshiyuki Itai

Research Interests

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Articles (13)

Body composition as a predictive factor for chemotherapy-induced peripheral neuropathy and dose-limiting toxicity in patients with endometrial cancer undergoing carboplatin and paclitaxel

Single-cell Multiome Characterizing Intercellular Communication and Intracellular Regulation of Epithelium and Mesenchymal during Secondary Palate Development in Mice

Radiation‐induced congenital malformations in Fukushima after the Fukushima Daiichi Nuclear Disaster

Investigating gene functions and single-cell expression profiles of de novo variants in orofacial clefts

De novo mutations disturb early brain development more frequently than common variants in schizophrenia

Publisher Correction: A novel NONO variant that causes developmental delay and cardiac phenotypes

A novel NONO variant that causes developmental delay and cardiac phenotypes

Skeletal anomaly and opisthotonus in early-onset epileptic encephalopathy with KCNQ2 abnormality.

De novo heterozygous variants in KIF5B cause kyphomelic dysplasia

Large-scale discovery of novel neurodevelopmental disorder-related genes through a unified analysis of single-nucleotide and copy number variants.

Progressive cerebral atrophies in three children with COL4A1 mutations.

Novel CLTC variants cause new brain and kidney phenotypes.

Cerebrovascular diseases in two patients with entire NSD1 deletion.

Collaborators (7)

Yoshihiro Watanabe

Atsushi Takata

Lukas Simon

yulin dai

Yohei Nitta

Jingchun Chen

Mitsuyoshi Suzuki

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