Un-Hwan Ha

Professor

Kangwon National University - Samcheok Campus

South Korea

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Un-Hwan Ha is a Professor at Korea University - Sejeong Campus in South Korea. His research primarily focuses on the virulence mechanisms of Pseudomonas aeruginosa and its interactions with host immune responses. Recent publications explore topics such as bacterial signaling pathways, lipopolysaccharide synthesis, and the role of effector proteins in modulating host cellular responses.

Articles (11)

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2025

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2025

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2025

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2025

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2023

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2023

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2022

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2021

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2021

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2020

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2020

Collaborators (2)

Hyun Sik Jun

SOUTH KOREA

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SUNG-JO KIM

Professor

Hoseo University

SOUTH KOREA

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Un-Hwan Ha

Research Interests

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About

Articles (11)

Pseudomonas aeruginosa lasR -deficient mutant contributes to bacterial virulence through enhancing the PhoB-mediated pathway in response to host environment

The fatty acid synthesis gene fabY affects lipopolysaccharide synthesis and colistin susceptibility in Pseudomonas aeruginosa

Elongation factor-G1A identified as a novel effector protein translocated into cells and a key modulator of Pseudomonas aeruginosa physiology and host cellular responses

Pseudomonas aeruginosa-derived DnaJ induces TLR2 expression through TLR10-mediated activation of the PI3K-SGK1 pathway in macrophages

DnaJ-induced miRNA-146a negatively regulates the expression of IL-8 in macrophages

Pseudomonas aeruginosa-Derived DnaJ Induces the Expression of IL−1β by Engaging the Interplay of p38 and ERK Signaling Pathways in Macrophages

DnaJ-induced TLR7 mediates an increase in interferons through the TLR4-engaged AKT/NF-κB and JNK signaling pathways in macrophages

The Anti-Tumor Effects of Oenothera odorata Extract Are Mediated by Inhibition of Glycolysis and Cellular Respiration in Cancer Cells

Pseudomonas aeruginosa DnaK Stimulates the Production of Pentraxin 3 via TLR4-Dependent NF-κB and ERK Signaling Pathways

HSP70-Homolog DnaK of Pseudomonas aeruginosa Increases the Production of IL-27 through Expression of EBI3 via TLR4-Dependent NF-κB and TLR4-Independent Akt Signaling

Maackiain, a compound derived from Sophora flavescens, increases IL‐1β production by amplifying nigericin‐mediated inflammasome activation

Collaborators (2)

Hyun Sik Jun

SUNG-JO KIM

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