Vicente Felipo

Head

Centro de Investigacion Principe Felipe

Spain

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About

Dr. Vicente Felipo is the Head of the Centro de Investigacion Principe Felipe in Spain. His research primarily focuses on neuroinflammation, cognitive impairment, and the effects of extracellular vesicles on brain health. Recent publications include studies on the pathological effects of bacterial extracellular vesicles and their therapeutic possibilities, as well as the role of peripheral inflammation in hepatic encephalopathy. Dr. Felipo's work contributes significantly to understanding the neurobiological mechanisms underlying liver disease and cognitive decline.

Articles (13)

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2025

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2023

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2023

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SPAIN

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Vicente Felipo

Research Interests

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Articles (13)

Extracellular vesicles from Lacticaseibacillus paracasei reduce neuroinflammation in hippocampus and restore some cognitive functions in hyperammonemic rats

Bacterial extracellular vesicles in the brain: Pathological effects and therapeutic possibilities

Role of peripheral inflammation in minimal hepatic encephalopathy

Increased levels and activation of the IL-17 receptor in microglia contribute to enhanced neuroinflammation in cerebellum of hyperammonemic rats

Golexanolone improves fatigue, motor incoordination and gait and memory in rats with bile duct ligation

Extracellular vesicles from mesenchymal stem cells improve neuroinflammation and neurotransmission in hippocampus and cognitive impairment in rats with mild liver damage and minimal hepatic encephalopathy

Mild Cognitive Impairment Is Associated with Enhanced Activation of Th17 Lymphocytes in Non-Alcoholic Fatty Liver Disease

Enhanced Activation of the S1PR2-IL-1β-Src-BDNF-TrkB Pathway Mediates Neuroinflammation in the Hippocampus and Cognitive Impairment in Hyperammonemic Rats

Patients who died with steatohepatitis or liver cirrhosis show neuroinflammation and neuronal loss in hippocampus

Sustained Hyperammonemia Activates NF-κB in Purkinje Neurons Through Activation of the TrkB-PI3K-AKT Pathway by Microglia-Derived BDNF in a Rat Model of Minimal Hepatic Encephalopathy

Extracellular vesicles from hyperammonemic rats induce neuroinflammation in hippocampus and impair cognition in control rats

Golexanolone, a GABAA receptor modulating steroid antagonist, restores motor coordination and cognitive function in hyperammonemic rats by dual effects on peripheral inflammation and neuroinflammation

Enhanced BDNF and TrkB Activation Enhance GABA Neurotransmission in Cerebellum in Hyperammonemia

Collaborators (5)

Salvador Benlloch

Amparo Urios

Gaspar Perez Martinez

Teresa San-Miguel

Carmina Montoliu

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