Wei Tong

Professor

University of Pennsylvania Perelman School of Medicine

United States

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Wei Tong is a Professor at the University of Pennsylvania Perelman School of Medicine in the United States. His research areas include myeloproliferative neoplasms, hematopoietic stem cell biology, and the molecular mechanisms underlying cancer progression. Recent articles highlight his work on NRASQ61R mutations, LNK/SH2B3's role in leukemia, and the effects of adipogenic lineage precursors on bone marrow repair.

Articles (13)

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2025

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2023

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2021

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2021

Collaborators (6)

Nathaniel Dyment

University of Pennsylvania

UNITED STATES

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Jun Gui

University of Pennsylvania

UNITED STATES

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Shuying Yang

Associate Professor

University of Pennsylvania

UNITED STATES

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Yulong Wei

University of Pennsylvania

UNITED STATES

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Michael Gonzalez

University of Pennsylvania

UNITED STATES

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Ziqing Li

University of Pennsylvania

UNITED STATES

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Wei Tong

Research Interests

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Articles (13)

Positive feedback loop between NRASQ61R mutation and RAB27B expression in endothelial cells

LNK/SH2B3 Loss Exacerbates the Development of Myeloproliferative Neoplasms in CBL-deficient Mice.

Hidden conformational codes

BRAF-V600E utilizes posttranscriptional mechanisms to amplify LPS-induced TNFα production in dendritic cells in a mouse model of Langerhans cell histiocytosis.

Transient expansion and myofibroblast conversion of adipogenic lineage precursors mediate bone marrow repair after radiation.

LNK (SH2B3) inhibition expands healthy and Fanconi anemia human hematopoietic stem and progenitor cells

Waking up CML leukemia stem cells for the kill

Bone marrow adipogenic lineage precursors promote osteoclastogenesis in bone remodeling and pathologic bone loss.

Depalmitoylation Rewires FLT3-ITD Signaling and Exacerbates Leukemia Progression.

RUNX-1 haploinsufficiency causes a marked deficiency of megakaryocyte-biased hematopoietic progenitor cells.

<i>Pparγ1</i> Facilitates ErbB2-Mammary Adenocarcinoma in Mice

HectD1 controls hematopoietic stem cell regeneration by coordinating ribosome assembly and protein synthesis.

Bone marrow adipogenic lineage precursors promote osteoclastogenesis in bone remodeling and pathologic bone loss

Collaborators (6)

Nathaniel Dyment

Jun Gui

Shuying Yang

Yulong Wei

Michael Gonzalez

Ziqing Li

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