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Wen Pan

Professor

University of Science and Technology of China

China

Research Interests

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Professors in Immunology

Articles (6)

Year:

2025

Year:

2024

Year:

2024

Year:

2024

Year:

2024

Year:

2024

Collaborators (1)

Shu Zhu

Dean

University of Science and Technology of China

CHINA

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Wen Pan

Research Interests

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Articles (6)

A B cell-IgA-epithelial axis enhances antitumor immunity and improves outcome in HPV-associated penile squamous cell carcinoma

Enterococcus-derived tyramine hijacks α2A-adrenergic receptor in intestinal stem cells to exacerbate colitis

DHX9 maintains epithelial homeostasis by restraining R-loop-mediated genomic instability in intestinal stem cells

Deficiency of immunoglobulin IgSF6 enhances antibacterial effects by promoting endoplasmic reticulum stress and the inflammatory response in intestinal macrophages

Bile acids modified by the intestinal microbiota promote colorectal cancer growth by suppressing CD8<sup>+</sup> T cell effector functions.

METTL9-catalyzed histidine methylation of S100A9 suppresses the anti-Staphylococcus aureus activity of neutrophils

Collaborators (1)

Shu Zhu

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Wen Pan

Research Interests

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Articles (6)

A B cell-IgA-epithelial axis enhances antitumor immunity and improves outcome in HPV-associated penile squamous cell carcinoma

Enterococcus-derived tyramine hijacks α2A-adrenergic receptor in intestinal stem cells to exacerbate colitis

DHX9 maintains epithelial homeostasis by restraining R-loop-mediated genomic instability in intestinal stem cells

Deficiency of immunoglobulin IgSF6 enhances antibacterial effects by promoting endoplasmic reticulum stress and the inflammatory response in intestinal macrophages

Bile acids modified by the intestinal microbiota promote colorectal cancer growth by suppressing CD8&lt;sup&gt;+&lt;/sup&gt; T cell effector functions.

METTL9-catalyzed histidine methylation of S100A9 suppresses the anti-Staphylococcus aureus activity of neutrophils

Collaborators (1)

Shu Zhu

Make your next move.

Bile acids modified by the intestinal microbiota promote colorectal cancer growth by suppressing CD8<sup>+</sup> T cell effector functions.