William Fagg

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Tenure Track Assistant Professor

University of Texas Medical Branch

United States

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About

William Fagg is a Tenure Track Assistant Professor at the University of Texas Medical Branch, USA. His research primarily focuses on regenerative medicine, with recent studies exploring the therapeutic applications of cell-free amniotic fluid, liver fibrosis, and epithelial-to-mesenchymal transition. He is also involved in investigating the molecular mechanisms underlying cardiac cell fate and the efficacy of novel treatments for non-healing wounds.

Recent Grants

Grant: Close

UCSC CIRM Training Program in Systems Biology of Stem cells

Open Date: 2009-10-01

Close Date: 2015-12-31

Grant: Close

Training Program in Molecular, Cell, and Developmental Biology

Open Date: 1999-07-01

Close Date: 2019-06-30

Articles (11)

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2025

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2025

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2024

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2022

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2022

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2022

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2022

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2021

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2021

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2020

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2020

Collaborators (4)

Omar Saldarriaga

Assistant Professor

University of Texas Medical Branch at Galveston

UNITED STATES

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Ricardo Rajsbaum

Associate PRofessor and Director of CVHII

Rutgers University

UNITED STATES

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Dr. Cassandra Modahl

Lecturer

Liverpool School of Tropical Medicine

UNITED KINGDOM

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Mariano A. Garcia-Blanco

University of Virginia

UNITED STATES

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William Fagg

Research Interests

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About

Recent Grants

Articles (11)

Amniotic fluid reduces liver fibrosis by attenuating hepatic stellate cell activation

Assessing the Safety, Tolerability and Efficacy of Cell‐Free Amniotic Fluid in the Treatment of Non‐Healing Venous Ulcers: Initial Experience From a Prospective, Multicenter, Phase II Study

Topical application of beta-blockers accelerates epithelialization to mesh skin grafted full-thickness burn in sheep

Cell-Free Amniotic Fluid and Regenerative Medicine: Current Applications and Future Opportunities

Comparative Analysis of Co-Cultured Amniotic Cell-Conditioned Media with Cell-Free Amniotic Fluid Reveals Differential Effects on Epithelial–Mesenchymal Transition and Myofibroblast Activation

Definition of germ layer cell lineage alternative splicing programs reveals a critical role for Quaking in specifying cardiac cell fate

Safety and efficacy of acellular human amniotic fluid and membrane in the treatment of non-healing wounds in a patient with chronic venous insufficiency

The RNA binding protein Quaking represses splicing of the Fibronectin EDA exon and downregulates the interferon response

Endoderm and Hepatic Progenitor Cells Engraft in the Quiescent Liver Concurrent with Intrinsically Activated Epithelial-to-Mesenchymal Transition

Topoisomerase III-β is required for efficient replication of positive-sense RNA viruses

The RNA binding protein Quaking represses host interferon response by downregulating MAVS

Collaborators (4)

Omar Saldarriaga

Ricardo Rajsbaum

Dr. Cassandra Modahl

Mariano A. Garcia-Blanco

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