Woo Sik Chung

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Gyeongsang National University

South Korea

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About

Woo Sik Chung is a Professor at Gyeongsang National University, South Korea. His research focuses on plant molecular biology, specifically the mechanisms of protein homeostasis, flowering regulation, and pathogen resistance in Arabidopsis. Recent articles include studies on the unfolded protein response, effects of 2,4-Diacetylphloroglucinol on induced systemic resistance, and the role of mitogen-activated protein kinases in plant signaling pathways.

Recent Grants

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Understanding of signaling pathways inhibiting flower development by environmental stresses

Open Date: 2016-06-01

Close Date: 2019-05-01

Grant: Close

Identification of plant immune induction pathways and molecules

Open Date: 2015-01-01

Close Date: 2017-12-01

Grant: Close

Identification of physiological function and action mechanism of new MAP kinase substrate proteins in Arabidopsis

Open Date: 2013-06-01

Close Date: 2016-05-01

Articles (12)

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2025

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2025

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2025

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2024

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2024

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2024

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2023

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2023

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2022

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2022

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2022

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2021

Collaborators (3)

Nayoung Lee

Gyeongsang National University

SOUTH KOREA

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Takato Imaizumi

UNITED STATES

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JUNGHOON PARK

Konkuk University

SOUTH KOREA

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Woo Sik Chung

Research Interests

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About

Recent Grants

Articles (12)

IRE1 coordinates UPR and ERAD pathways to maintain protein homeostasis under ER stress in Arabidopsis

The bZIP3‐AS1 complex promotes CONSTANS‐induced FLOWERING LOCUS T activation in a daylength‐dependent manner

2,4-Diacetylphloroglucinol (DAPG) Enhances Induced Systemic Resistance Against Necrotrophic Pathogens Through the COI1-Mediated JA Signaling Pathway in Arabidopsis

Identification of mitogen-activated protein kinases substrates in Arabidopsis using kinase client assay

ABA INSENSITIVE 2 promotes flowering by inhibiting OST1/ABI5-dependent FLOWERING LOCUS C transcription in Arabidopsis

Proteomic analysis of Arabidopsis leaves in response to 2,4-diacetylphloroglucinol (DAPG)

The role of protein phosphatase 2A (PP2A) in the unfolded protein response (UPR) of plants

Quercetin induces pathogen resistance through the increase of salicylic acid biosynthesis in Arabidopsis

Phosphorylation of the auxin signaling transcriptional repressor IAA15 by MPKs is required for the suppression of root development under drought stress in Arabidopsis

Ca2+/CaM increases the necrotrophic pathogen resistance through the inhibition of a CaM-regulated dual-specificity protein phosphatase 1 in Arabidopsis

Ca2+/Calmodulin Activates an MAP Kinase Through the Inhibition of a Protein Phosphatase (DsPTP1) in Arabidopsis

Naringenin Induces Pathogen Resistance Against Pseudomonas syringae Through the Activation of NPR1 in Arabidopsis

Collaborators (3)

Nayoung Lee

Takato Imaizumi

JUNGHOON PARK

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