xinbin chen

Professor

University of California, Davis

United States

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Professor Xinbin Chen is a faculty member at the University of California Davis in the United States. His research focuses on tumor biology, specifically the roles of various p53 isoforms in tumor suppression and cancer treatment. Recent articles authored by Professor Chen explore critical pathways and molecules involved in tumorigenesis, lipid homeostasis, and the modulation of cellular growth processes.

Articles (18)

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2025

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2022

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2022

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2022

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2021

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2021

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2021

Collaborators (5)

Paramita Ghosh

Professor

University of California Davis School of Medicine

UNITED STATES

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Luke Wittenburg

UNITED STATES

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Jared Shaw

Professor

University of California, Davis

UNITED STATES

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Christopher Lucchesi

University of California, Davis

UNITED STATES

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jin zhang

University of California, Davis

UNITED STATES

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xinbin chen

Research Interests

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Articles (18)

ANGEL2 Modulates Wild-type TP53 Translation and Doxorubicin Chemosensitivity in Colon Cancer

Development of Novel Peptides That Target the Ninjurin 1 and 2 Pathways to Inhibit Cell Growth and Survival via p53

TAp63γ is the primary isoform of TP63 for tumor suppression but not development

Correction: Isoform-specific disruption of the TP73 gene reveals a critical role for TAp73γ in tumorigenesis via leptin

Ferredoxin 2 Is Critical for Tumor Suppression and Lipid Homeostasis but Dispensable for Embryonic Development

Ninjurin 2, a Cell Adhesion Molecule and a Target of p53, Modulates Wild-Type p53 in Growth Suppression and Mutant p53 in Growth Promotion

Ninjurin 2 Modulates Tumorigenesis, Inflammation, and Metabolism via Pyroptosis

Ferredoxin 1 is essential for embryonic development and lipid homeostasis

Identification of a First-in-Class Small-Molecule Inhibitor of the EIF4E-RBM38 Complex That Enhances Wild-type TP53 Protein Translation for Tumor Growth Suppression

Isoform-specific disruption of the TP73 gene reveals a critical role for TAp73γ in tumorigenesis via leptin

TP73 Isoform-specific disruption reveals a critical role of TAp73beta in growth suppression and inflammatory response

Microglia-Derived Olfactomedin-like 3 Is a Potent Angiogenic Factor in Primary Mouse Brain Endothelial Cells: A Novel Target for Glioblastoma

ZFP14 Regulates Cancer Cell Growth and Migration by Modulating p53 Protein Stability as Part of the MDM2 E3 Ubiquitin Ligase Complex

p73α1, an Isoform of the p73 Tumor Suppressor, Modulates Lipid Metabolism and Cancer Cell Growth via Stearoyl-CoA Desaturase-1

Ferredoxin reductase and p53 are necessary for lipid homeostasis and tumor suppression through the ABCA1–SREBP pathway

Microglia-Derived Olfactomedin-like 3 Promotes Pro-Tumorigenic Microglial Function and Malignant Features of Glioma Cells

Mice Deficient in the RNA-Binding Protein Zfp871 Are Prone to Early Death and Steatohepatitis in Part through the p53–Mdm2 Axis

Small Proline-Rich Protein 2A and 2D Are Regulated by the RBM38-p73 Axis and Associated with p73-Dependent Suppression of Chronic Inflammation

Collaborators (5)

Paramita Ghosh

Luke Wittenburg

Jared Shaw

Christopher Lucchesi

jin zhang

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