Yi Sun

Professor Emeritus

University of Michigan

United States

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About

Yi Sun is a Professor Emeritus at the University of Michigan, USA. His research primarily focuses on the mechanisms of cancer development and therapy, with recent studies examining the roles of E3 ligases in tumorigenesis and cancer metabolism. Dr. Sun's work includes significant contributions to understanding the pathways involved in lymphomagenesis, colon cancer, and liver tumorigenesis.

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Yi Sun

Research Interests

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About

Articles (25)

DEPTOR suppresses lymphomagenesis by promoting EGFR degradation via HUWE1 E3 ligase

SMURF1: a promising target for colon cancer therapy

RHEB neddylation by the UBE2F-SAG axis enhances mTORC1 activity and aggravates liver tumorigenesis

E3 Ligase Rbx1 Orchestrates Thymus Development and Fate Determination of αβ-γδ T Cells

Highly activated neddylation pathway in lung cancer and chemical targeted intervention,蛋白质的拟素化通路在肺癌中的激活和化学靶向干预

Proteome-Wide Data Guides the Discovery of Lysine-Targeting Covalent Inhibitors Using DNA-Encoded Chemical Libraries

The TRIM22-CDT2 axis is the key mediator of the p53-Rb signals in growth control of HPV-positive cervical carcinoma cells

USP10 deubiquitylates and stabilizes DIRAS2 to suppress the growth of pancreatic cancer cells

Sag deletion promotes DMBA/TPA‐induced skin carcinogenesis via YAP accumulation

The CRL3 KCTD10 ubiquitin ligase–USP18 axis coordinately regulates cystine uptake and ferroptosis by modulating SLC7A11

SHOC2 plays an oncogenic or tumor-suppressive role by differentially targeting the MAPK and mTORC1 signals in liver cancer

The UBE2F-CRL5ASB11-DIRAS2 axis is an oncogene and tumor suppressor cascade in pancreatic cancer cells

Protein neddylation and its role in health and diseases

Neddylation is a novel therapeutic target for lupus by regulating double negative T cell homeostasis

CRL3 E3 ligase regulates glutamine and cystine metabolisms

The TRAF2-p62 axis promotes proliferation and survival of liver cancer by activating mTORC1 pathway

Neddylation‐CRLs regulate the functions of Treg immune cells

The Functional Redundancy of Neddylation E2s and E3s in Modulating the Fitness of Regulatory T Cells

Early growth response-1 is a new substrate of the GSK3β-FBXW7 axis

The Sag-Shoc2 axis regulates conversion of mPanINs to cystic lesions in Kras pancreatic tumor model

PD-L1 induction via the MEK-JNK-AP1 axis by a neddylation inhibitor promotes cancer-associated immunosuppression

The Ube2m-Rbx1 neddylation-Cullin-RING-Ligase proteins are essential for the maintenance of Regulatory T cell fitness

Discovery of a small molecule inhibitor of cullin neddylation that triggers ER stress to induce autophagy

USP2 is an SKP2 deubiquitylase that stabilizes both SKP2 and its substrates

Mutual inactivation of two RING-finger-type E3 ubiquitin ligases during cell cycle progression

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