Yiliang Chen

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Assistant Professor

Medical College of Wisconsin

United States

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Recent Grants

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Role of TLR9 and mitochondrial DNA in regulating microvascular and immune cell dysfunction post-COVID-19

Open Date: 2022-04-01

Close Date: 2025-03-01

Grant: Close

Pim1 kinase coordinates PPAR gamma pathway and mitochondrial function to mediate pro-atherogenic responses in macrophages

Open Date: 2021-04-01

Close Date: 2025-03-01

Grant: Close

Macrophage immunometabolism and mitochondrial functions

Open Date: 2020-09-01

Close Date: 2023-08-01

Grant: Close

Na/K-ATPase-mediated signaling in the development of atherosclerosis

Open Date: 2017-07-01

Close Date: 2020-06-01

Grant: Close

CD36/Na/K-ATPase Signaling Complex in Macrophage Cholesterol Metabolism and Atherosclerois

Open Date: 2013-01-01

Close Date: 2014-12-01

Articles (12)

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2024

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2024

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2023

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2022

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2022

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2022

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2021

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2021

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2021

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2021

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2020

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2020

Collaborators (3)

Liquan Cai

Marshall University

UNITED STATES

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Weiguo Cui

UNITED STATES

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Sneha S. Pillai

Marshall University

UNITED STATES

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Yiliang Chen

Research Interests

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Recent Grants

Articles (12)

Role of Na/K-ATPase α1 caveolin-binding motif in adipogenesis

Targeting mitochondrial dynamics and redox regulation in cardiovascular diseases

Na/K-ATPase signaling tonically inhibits sodium reabsorption in the renal proximal tubule.

A CD36 transmembrane domain peptide interrupts CD36 interactions with membrane partners on macrophages and inhibits atherogenic functions.

Na/K-ATPase suppresses LPS-induced pro-inflammatory signaling through Lyn.

CD36, a signaling receptor and fatty acid transporter that regulates immune cell metabolism and fate

AMPK-deficiency forces metformin-challenged cancer cells to switch from carbohydrate metabolism to ketogenesis to support energy metabolism

Hypertriglyceridemia during hospitalization independently associates with mortality in patients with COVID-19

CD36 promotes NLRP3 inflammasome activation via the mtROS pathway in renal tubular epithelial cells of diabetic kidneys.

Targeting PIM1-Mediated Metabolism in Myeloid Suppressor Cells to Treat Cancer.

Regulation of Na/K-ATPase expression by cholesterol: isoform specificity and the molecular mechanism.

A caveolin binding motif in Na/K-ATPase is required for stem cell differentiation and organogenesis in mammals and C.elegans

Collaborators (3)

Liquan Cai

Weiguo Cui

Sneha S. Pillai

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