Yoki Nakamura

Assistant Professor

Hiroshima University

Japan

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Yoki Nakamura is an Assistant Professor at Hiroshima University in Japan. His research focuses on neurobiology, particularly the roles of microglial receptors in mitochondrial function and energy metabolism, as well as the effects of nerve injuries on pain behaviors. Recent publications highlight his investigations into cognitive impairment related to microglial activation and the biological evaluation of phototherapeutic compounds.

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2025

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2022

Collaborators (4)

Yutaka Kikuchi

Professor

Hiroshima University

JAPAN

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Kouichi Hashimoto

Hiroshima University

JAPAN

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Manabu Abe

Hiroshima University

JAPAN

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Satoshi Asano

Hiroshima University

JAPAN

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Yoki Nakamura

Research Interests

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Articles (20)

Preventive effect of RAGE antagonists on distal infraorbital nerve injury-induced pain behaviors of male and female mice

Microglial α7‐Nicotinic Acetylcholine Receptors Are Expressed in Mitochondria Rather Than on the Plasma Membrane: Roles in Mitochondrial Function

Microglial α7-Nicotinic Acetylcholine Receptors Are Expressed in Mitochondria Rather Than on the Plasma Membrane: Roles in Mitochondrial Function

Age-related Dysregulation of Energy Metabolism and Mitochondrial Dynamics in Microglia

Age-related Dysregulation of Energy Metabolism and Mitochondrial Dynamics in Microglia

Design, Synthesis, and Biological Evaluation of BODIPY-Caged Resiquimod as a Dual-Acting Phototherapeutic

Design, Synthesis, and Biological Evaluation of BODIPY-Caged Resiquimod as a Dual-Acting Phototherapeutic

Hippocampal microglial activation induces cognitive impairment and allodynia through neuronal plasticity changes in male mice with neuropathic pain

Hippocampal microglial activation induces cognitive impairment and allodynia through neuronal plasticity changes in male mice with neuropathic pain

Ketamine-induced pica behavior as nausea and vomiting involves dopamine and serotonin receptor activation in the area postrema of rats: A comparison with etomidate and pentobarbital

Inhibition of nuclear receptor RORγ ameliorates mechanical hypersensitivity through the suppression of spinal microglial activation

Mitochondrial dysfunction and type I interferon signaling induce anxiodepressive-like behaviors in mice with neuropathic pain

Evaluation of intracellular signal molecules that regulate TLR4-stimulated inflammatory mediator expression in cultured rat chondrocytes

Blockade of receptor for advanced glycation end-products with azeliragon ameliorates streptozotocin-induced diabetic neuropathy

Microglia and innate immunity in Alzheimer’s disease

High-mobility group box 1-mediated hippocampal microglial activation induces cognitive impairment in mice with neuropathic pain.

Prenatal exposure to valproic acid causes allodynia associated with spinal microglial activation

Anti-interleukin-6 receptor antibody improves allodynia and cognitive impairment in mice with neuropathic pain following partial sciatic nerve ligation

Downregulation of connexin 43 potentiates amitriptyline-induced brain-derived neurotrophic factor expression in primary astrocytes through lysophosphatidic acid receptor1/3, Src, and extracellular signal-regulated kinase

Mirogabalin alleviates nociceptive hypersensitivity without causing sedation in a mouse model of post-traumatic trigeminal neuropathy

Collaborators (4)

Yutaka Kikuchi

Kouichi Hashimoto

Manabu Abe

Satoshi Asano

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