Yong-Hyun Han

Professor

Changwon National University

South Korea

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Yong-Hyun Han is a Professor at Kangwon National University in South Korea. His research primarily focuses on liver diseases, immunosenescence, and metabolic disorders, as evidenced by recent publications exploring the effects of biologics on fibrotic progression and the underlying mechanisms of nonalcoholic steatohepatitis. He investigates the roles of various cytokines and macrophages in liver pathology, contributing valuable insights into the treatment and understanding of age-related and metabolic diseases.

Articles (15)

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2025

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2023

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2022

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2022

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2021

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2021

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2021

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2021

Collaborators (2)

Brad W. Warner

Washington University School of Medicine

UNITED STATES

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Hyun-Jeong Ko

Professor

Changwon National University

SOUTH KOREA

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Yong-Hyun Han

Research Interests

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Articles (15)

Treatment of IL-18-binding protein biologics suppresses fibrotic progression in metabolic dysfunction-associated steatohepatitis

Immunosenescence and age-related immune cells: causes of age-related diseases

The Root Extract of <i>Rosa multiflora</i> Ameliorates Nonalcoholic Steatohepatitis Development via Blockade of De Novo Lipogenesis and Inflammation

Panaxydol extracted from Panax ginseng inhibits NLRP3 inflammasome activation to ameliorate NASH-induced liver injury

Hepatic TREM2+ macrophages express matrix metalloproteinases to control fibrotic scar formation.

Disruption of IL-18 signaling via engineered IL-18BP biologics alleviates experimental cholestatic liver disease

Lubiprostone significantly represses fatty liver diseases via induction of mucin and HDL release in mice.

Chemotactic cytokines secreted from Kupffer cells contribute to the sex-dependent susceptibility to non-alcoholic fatty liver diseases in mice.

Lipid absorption and overall intestinal lymphatic transport are impaired following partial small bowel resection in mice.

Roles of heterogenous hepatic macrophages in the progression of liver diseases

Loss of ERdj5 exacerbates oxidative stress in mice with alcoholic liver disease via suppressing Nrf2.

RORα contributes to the maintenance of genome ploidy in the liver of mice with diet-induced nonalcoholic steatohepatitis.

Ileitis-associated tertiary lymphoid organs arise at lymphatic valves and impede mesenteric lymph flow in response to tumor necrosis factor.

Specialized Proresolving Mediators for Therapeutic Interventions Targeting Metabolic and Inflammatory Disorders.

RORα Enhances Lysosomal Acidification and Autophagic Flux in the Hepatocytes.

Collaborators (2)

Brad W. Warner

Hyun-Jeong Ko

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