Yoshiko Murakami

SA professor

Osaka University

Japan

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Yoshiko Murakami is an Associate Professor at Osaka University in Japan. Her research focuses on glycosylphosphatidylinositol (GPI) biosynthesis, the mechanisms underlying inherited GPI deficiencies, and their implications for various diseases. Recent articles include studies on therapeutic potentials of gene therapy for GPI deficiencies and insights from genome-wide CRISPR screens related to lipid scramblases. She also investigates the cross-talk between GPI biosynthesis and other lipid pathways.

Articles (10)

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2023

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2022

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2022

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2022

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2022

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2021

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2021

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2021

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2020

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2020

Collaborators (8)

Anant Kumar Menon

Professor

Weill Cornell Medicine

UNITED STATES

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Yugo Iwasaki

Chubu University

JAPAN

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Vincent Cantagrel

Université Paris Cité

FRANCE

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Daniel Varon Silva

Professor of Organic and Bioorganic Chemistry

Hochschule für Life Sciences FHNW

SWITZERLAND

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Daisuke Kobayashi

Health Sciences University of Hokkaido

JAPAN

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Yuta Maki

Osaka University

JAPAN

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Slim Azouzi

FRANCE

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Keiichiro Suzuki

Osaka University

JAPAN

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Yoshiko Murakami

Research Interests

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Articles (10)

Accumulated precursors of specific GPI-anchored proteins upregulate GPI biosynthesis with ARV1

Sequential hydrolysis of FAD by ecto-5′ nucleotidase CD73 and alkaline phosphatase is required for uptake of vitamin B2 into cells

Genome-wide CRISPR screen reveals CLPTM1L as a lipid scramblase required for efficient glycosylphosphatidylinositol biosynthesis

Ethanolamine‐phosphate on the second mannose is a preferential bridge for some GPI‐anchored proteins

Establishment of mouse model of inherited PIGO deficiency and therapeutic potential of AAV-based gene therapy

Rescue of Glycosylphosphatidylinositol-Anchored Protein Biosynthesis Using Synthetic Glycosylphosphatidylinositol Oligosaccharides

Inherited glycosylphosphatidylinositol defects cause the rare Emm-negative blood phenotype and developmental disorders.

PIGG variant pathogenicity assessment reveals characteristic features within 19 families.

Paroxysmal nocturnal hemoglobinuria caused by CN-LOH of constitutional PIGB mutation and 70-kbp microdeletion on 15q.

Cross-talks of glycosylphosphatidylinositol biosynthesis with glycosphingolipid biosynthesis and ER-associated degradation.

Collaborators (8)

Anant Kumar Menon

Yugo Iwasaki

Vincent Cantagrel

Daniel Varon Silva

Daisuke Kobayashi

Yuta Maki

Slim Azouzi

Keiichiro Suzuki

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