Yoshinobu Saito

Assistant Professor

Osaka University

Japan

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Yoshinobu Saito is an Assistant Professor at Osaka University, Japan. His research primarily focuses on liver cancer biology, apoptosis, and the role of cellular microenvironments in hepatic functions. Recent publications highlight his work on hepatocyte apoptosis, liver zonation, and biomarkers for liver cancer in patients with metabolic diseases. He is also investigating the roles of different subpopulations of hepatic stellate cells in hepatocarcinogenesis and their impact on cancer prognosis.

Articles (12)

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2025

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2025

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2024

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2024

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2023

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2022

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2022

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2022

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2021

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2021

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2021

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2021

Collaborators (9)

Atsushi Sugimoto

Columbia University in the City of New York

UNITED STATES

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Ira Tabas

Professor

Columbia University Irving Medical Center

UNITED STATES

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Moritz Peiseler

Charité - Universitätsmedizin Berlin

GERMANY

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Takahiro Kodama

Assistant Professor

Osaka University

JAPAN

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Nicholas Arpaia

Assistant Professor

Columbia University Irving Medical Center

UNITED STATES

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Raghu Kalluri

Professor and Chairman

The University of Texas MD Anderson Cancer Center

UNITED STATES

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Utpal Pajvani

UNITED STATES

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Xiaobo Wang

Assistant Professor

Columbia University Irving Medical Center

UNITED STATES

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Naoto Fujiwara

Mie University Hospital

JAPAN

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Yoshinobu Saito

Research Interests

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Articles (12)

Collaborative orchestration of BH3-only proteins governs Bak/Bax-dependent hepatocyte apoptosis under antiapoptotic protein-deficiency in mice

Hepatic stellate cells control liver zonation, size and functions via R-spondin 3

Serum growth differentiation factor 15 is a novel biomarker with high predictive capability for liver cancer occurrence in patients with MASLD regardless of liver fibrosis

The serum tenascin C level is a marker of metabolic disorder-related inflammation affecting pancreatic cancer prognosis

A Therapeutically Targetable TAZ-TEAD2 Pathway Drives the Growth of Hepatocellular Carcinoma via ANLN and KIF23

Opposing roles of hepatic stellate cell subpopulations in hepatocarcinogenesis

TAZ-induced Cybb contributes to liver tumor formation in non-alcoholic steatohepatitis

Activation of p53 After Irradiation Impairs the Regenerative Capacity of the Mouse Liver

Promotion of cholangiocarcinoma growth by diverse cancer-associated fibroblast subpopulations

Gab1 in livers with persistent hepatocyte apoptosis has an antiapoptotic effect and reduces chronic liver injury, fibrosis, and tumorigenesis.

Gab1 in livers with persistent hepatocyte apoptosis has an antiapoptotic effect and reduces chronic liver injury, fibrosis, and tumorigenesis

Persistent hepatocyte apoptosis promotes tumorigenesis from diethylnitrosamine-transformed hepatocytes through increased oxidative stress, independent of compensatory liver regeneration.

Collaborators (9)

Atsushi Sugimoto

Ira Tabas

Moritz Peiseler

Takahiro Kodama

Nicholas Arpaia

Raghu Kalluri

Utpal Pajvani

Xiaobo Wang

Naoto Fujiwara

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