Yuichi Akama

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Feinstein Institute for Medical Research

United States

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Recent Grants

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間質性肺炎急性増悪の新規メカニズム：エクソソーム型サイトカインによる線維化作用

Open Date: 2022-04-01

Close Date: 2025-03-31

Grant: Close

Study of the roles played by exosomal microRNAs of intestinal epithelial cells in downregulating septic inflammations

Open Date: 2022-04-01

Close Date: 2025-03-31

Grant: Close

自然リンパ球が骨格筋の機能的萎縮を抑止する:敗血症サバイバー早期離床への新規治療標的の研究

Open Date: 2021-11-01

Close Date: 2023-03-01

Grant: Close

ILC2の疲弊が惹起する代謝制御異常：敗血症性ミオパチーとPD-L1エキソソーム

Open Date: 2021-04-01

Close Date: 2024-03-31

Grant: Close

エキソソームPD-L1が敗血症性免疫麻痺を誘導するメカニズムの解明

Open Date: 2021-04-01

Close Date: 2024-03-31

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Ping Wang

Professor, Chief Scientific Officer

Feinstein Institute for Medical Research

UNITED STATES

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Yuichi Akama

Research Interests

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Recent Grants

Articles (17)

A novel molecule targeting neutrophil-mediated B-1a cell trogocytosis attenuates sepsis-induced acute lung injury.

CD300ld promotes neutrophil bacterial phagocytosis in sepsis.

Machine Learning-Based Prediction of Delirium and Risk Factor Identification in ICU Patients with Burns: A Retrospective Observational Study.

Extracellular CIRP induces CD4CD8αα intraepithelial lymphocyte cytotoxicity in sepsis

Roles of programmed death‐1 and muscle innate lymphoid cell‐derived interleukin 13 in sepsis‐induced intensive care unit‐acquired weakness

miR-200c-3p regulates α4 integrin-mediated T cell adhesion and migration

MicroRNA Profiles in Intestinal Epithelial Cells in a Mouse Model of Sepsis

Possible Metastatic Stage-Dependent ILC2 Activation Induces Differential Functions of MDSCs through IL-13/IL-13Rα1 Signaling during the Progression of Breast Cancer Lung Metastasis

Elevated Plasma Soluble PD-L1 Levels in Out-of-Hospital Cardiac Arrest Patients.

Cellular and Exosomal Regulations of Sepsis-Induced Metabolic Alterations.

Identification of a small pericardial effusion on contrast-enhanced computed tomography indicating cardiac perforation and pericardial injury following blunt trauma: A case report.

Sepsis Induces Deregulation of IL-13 Production and PD-1 Expression in Lung Group 2 Innate Lymphoid Cells.

Veno-arterial extracorporeal membrane oxygenation and targeted temperature management in tricyclic antidepressant-induced cardiac arrest: A case report and literature review.

The Lectin-Like Domain of Thrombomodulin Inhibits β1 Integrin-Dependent Binding of Human Breast Cancer-Derived Cell Lines to Fibronectin.

Sepsis Induces Deregulation of IL-13 Production and PD-1 Expression in Lung Group 2 Innate Lymphoid Cells

Intestinal Epithelium-Derived Luminally Released Extracellular Vesicles in Sepsis Exhibit the Ability to Suppress TNF-a and IL-17A Expression in Mucosal Inflammation.

The Role of Innate Lymphoid Cells in the Regulation of Immune Homeostasis in Sepsis-Mediated Lung Inflammation.

Collaborators (6)

Monowar Aziz

Ryota Sakamoto

Eun Jeong Park

Arong Gaowa

Motoshi Takao

Ping Wang

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