Zhe Ji

Assistant Professor

Northwestern University Feinberg School of Medicine

United States

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About

Zhe Ji is an Assistant Professor at Northwestern University Feinberg School of Medicine, USA. His research focuses on deep learning applications in polyadenylation, cancer biology, and the characterization of noncanonical peptides. Recent publications include studies on the molecular determinants of polyadenylation site usage, gene networks in cancer metastasis, and the role of novel proteins in immune responses.

Articles (15)

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2025

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2024

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2024

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2024

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2024

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2022

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2021

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2021

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2021

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2021

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2020

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2020

Collaborators (8)

Bo Li

Assistant Professor

Harvard Medical School

UNITED STATES

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Vincent LAVOUE

Université de Rennes 1

FRANCE

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patrick legembre

Director of Research (visiting professor)

Université de Limoges

FRANCE

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Constance Cepko

Professor

Harvard Medical School

UNITED STATES

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TINGYOU WANG

Northwestern University Feinberg School of Medicine

UNITED STATES

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Derek Wainwright

Associate Professor, Scientific Director for Brain Tumor Research

Loyola University Chicago Stritch School of Medicine

UNITED STATES

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Fahmin Basher

Duke University Medical Center

UNITED STATES

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Sui Wang

Assistant professor

Stanford University

UNITED STATES

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Zhe Ji

Research Interests

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About

Articles (15)

The Advances in Deep Learning Modeling of Polyadenylation Codes

Comparative analyses of gene networks mediating cancer metastatic potentials across lineage types

The prolyl isomerase FKBP11 is a secretory translocon accessory factor.

Delineating yeast cleavage and polyadenylation signals using deep learning

Widespread stable noncanonical peptides identified by integrated analyses of ribosome profiling and ORF features

Deep learning of human polyadenylation sites at nucleotide resolution reveals molecular determinants of site usage and relevance in disease

Low-input RNase footprinting for simultaneous quantification of cytosolic and mitochondrial translation.

Tumor-derived NKG2D ligand sMIC reprograms NK cells to an inflammatory phenotype through CBM signalosome activation

CD95/Fas protects triple negative breast cancer from anti-tumor activity of NK cells

Unannotated proteins expand the MHC-I-restricted immunopeptidome in cancer.

Spike-in normalization for single-cell RNA-seq reveals dynamic global transcriptional activity mediating anticancer drug response.

A PRC2-independent function for EZH2 in regulating rRNA 2'-O methylation and IRES-dependent translation.

Noncanonical open reading frames encode functional proteins essential for cancer cell survival.

Cell type- and stage-specific expression of Otx2 is regulated by multiple transcription factors and cis-regulatory modules in the retina

Antibody targeting tumor-derived soluble NKG2D ligand sMIC reprograms NK cell homeostatic survival and function and enhances melanoma response to PDL1 blockade therapy.

Collaborators (8)

Bo Li

Vincent LAVOUE

patrick legembre

Constance Cepko

TINGYOU WANG

Derek Wainwright

Fahmin Basher

Sui Wang

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