Abinaya Chandrasekaran

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Assistant Professor

University of Copenhagen

Denmark

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About

Abinaya Chandrasekaran is an Assistant Professor at the University of Copenhagen, Denmark. Her research focuses on neurodegenerative diseases, employing innovative techniques such as gene editing and human induced pluripotent stem cells (hiPSCs) to model conditions like Huntington’s disease and epilepsy. Recent publications highlight her work on the metabolic and inflammatory profiles of microglia in frontotemporal dementia and the generation of disease-specific models for Alzheimer’s and amyotrophic lateral sclerosis.

Recent Grants

Grant: Close

RhoA links with Alzheimers Diseases

Open Date: 2022-01-01

Close Date: 2024-01-01

Grant: Close

CACNA1A role in Epilepsy

Open Date: 2022-01-01

Close Date: 2022-01-01

Articles (10)

Year:

2024

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2024

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2024

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2023

Year:

2023

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2023

Year:

2023

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2021

Year:

2021

Year:

2020

Collaborators (11)

Jan Gorodkin

Professor

University of Copenhagen

DENMARK

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Charlotte Brasch Andersen

Asso. professor

University of Southern Denmark

DENMARK

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Mathew Blurton-Jones

Professor

University of California, Irvine

UNITED STATES

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Ernst Stefan Seemann

Københavns Universitet

DENMARK

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Hans H. Wandall

University of Copenhagen

DENMARK

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Nadezhda T. Doncheva

Københavns Universitet

DENMARK

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Giulia Ilaria Corsi

Københavns Universitet

DENMARK

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Kristine Freude

Associate Professor

University of Copenhagen

DENMARK

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Henriette Reventlow Frederiksen

University of Copenhagen

DENMARK

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Mette Berendt

Københavns Universitet

DENMARK

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Noortje de Haan

Københavns Universitet

DENMARK

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Abinaya Chandrasekaran

Research Interests

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About

Recent Grants

Articles (10)

Generation of three isogenic gene-edited Huntington’s disease human embryonic stem cell lines with DOX-inducible NGN2 expression cassette in the AAVS1 safe locus

Generation of two patient specific GABRD variants and their isogenic controls for modeling epilepsy

Generation of Human Induced Pluripotent Stem Cell (hiPSC)-Derived Astrocytes for Amyotrophic Lateral Sclerosis and Other Neurodegenerative Disease Studies

Alteration of microglial metabolism and inflammatory profile contributes to neurotoxicity in a hiPSC-derived microglia model of frontotemporal dementia 3.

Golgi fragmentation - One of the earliest organelle phenotypes in Alzheimer's disease neurons.

Generation of eight hiPSCs lines from two pathogenic variants in CACNA1A using the CRISPR-Cas9 gene editing technology

Charting the Explicit Path: Translational Dynamics of Hepatic Bioengineering from Experimental Benchmarks to Practical Bedside Applications

Neural Derivates of Canine Induced Pluripotent Stem Cells-Like Cells From a Mild Cognitive Impairment Dog.

Astrocytic reactivity triggered by defective autophagy and metabolic failure causes neurotoxicity in frontotemporal dementia type 3.

Glutamate-Glutamine Homeostasis is Perturbed in Neurons and Astrocytes derived from Patient iPSC Models of Frontotemporal Dementia

Collaborators (11)

Jan Gorodkin

Charlotte Brasch Andersen

Mathew Blurton-Jones

Ernst Stefan Seemann

Hans H. Wandall

Nadezhda T. Doncheva

Giulia Ilaria Corsi

Kristine Freude

Henriette Reventlow Frederiksen

Mette Berendt

Noortje de Haan

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