Aimo Kannt

Head of Department

Fraunhofer Institute for Translational Medicine and Pharmacology

Germany

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About

Aimo Kannt is the Head of Department at the Fraunhofer Institute for Translational Medicine and Pharmacology in Germany. His research areas encompass translational medicine, pharmacology, and immunology, with a focus on cytokine signaling, metabolic disorders, and the immune responses in diseases such as psoriasis and COVID-19. His recent publications include studies on macrophage and dendritic cell phenotypes, serum proteomics in acute liver failure, and novel therapeutic concepts targeting specific cellular pathways.

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2025

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2021

Collaborators (18)

Marta Afonso

Assistant Professor

Faculty of Pharmacy, University of Lisbon

PORTUGAL

Aimo Kannt

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Articles (20)

Serum proteomics of adults with Acute Liver Failure provide mechanistic insights and attractive prognostic biomarkers

High content imaging shows distinct macrophage and dendritic cell phenotypes for psoriasis and atopic dermatitis.

Papaverine Targets STAT Signaling: A Dual-Action Therapy Option Against SARS-CoV-2.

Serum Carnosinase 1 Is Not Associated with Insulin Resistance or Glucose Metabolism in a Type 1 Diabetes Cohort

Proteomic, metabolomic and lipidomic profiles in community acquired pneumonia for differentiating viral and bacterial infections.

An unbiased ranking of murine dietary models based on their proximity to human metabolic dysfunction-associated steatotic liver disease (MASLD)

Neutralizing IL-38 activates γδ T cell-dependent antitumor immunity and sensitizes for chemotherapy

Chemogenomic Screening in a Patient-Derived 3D Fatty Liver Disease Model Reveals the CHRM1-TRPM8 Axis as a Novel Module for Targeted Intervention.

Identification of CRTH2 as a New PPARγ-Target Gene in T Cells Suggested CRTH2 Dependent Conversion of Th2 Cells as Therapeutic Concept in COVID-19 Infection

Serum carnosinase 1, an early indicator for incident microalbuminuria in type 1 diabetes.

Association of Dietary Patterns with MRI Markers of Hepatic Inflammation and Fibrosis in the MAST4HEALTH Study

The blood-brain barrier is dysregulated in COVID-19 and serves as a CNS entry route for SARS-CoV-2.

Novel tricyclic small molecule inhibitors of Nicotinamide N-methyltransferase for the treatment of metabolic disorders.

Analysis of 16 studies in nine rodent models does not support the hypothesis that diabetic polyuria is a main reason of urinary bladder enlargement.

Association Between Serum Carnosinase Concentration and Activity and Renal Function Impairment in a Type-2 Diabetes Cohort.

Ibuprofen, Flurbiprofen, Etoricoxib or Paracetamol Do Not Influence ACE2 Expression and Activity In Vitro or in Mice and Do Not Exacerbate In-Vitro SARS-CoV-2 Infection

Nutrigenetic Interactions Might Modulate the Antioxidant and Anti-Inflammatory Status in Mastiha-Supplemented Patients With NAFLD

Mastiha has efficacy in immune-mediated inflammatory diseases through a microRNA-155 Th17 dependent action

Expanding the arsenal of E3 ubiquitin ligases for proximity-induced protein degradation

Activation of thyroid hormone receptor‐β improved disease activity and metabolism independent of body weight in a mouse model of non‐alcoholic steatohepatitis and fibrosis

Collaborators (18)

Marta Afonso

Francisco Jose Roig Molina

Andreas J. Hülsmeier

Martin von Bergen

George Dedoussis

Fiona Oakley

Manuel A Friese

Ann Driessen

Jochen Bodem

Ioanna Panagiota Kalafati

Elisabeth Adam

Petra Tomanová

CHARALAMPIA AMERIKANOU

Diego Sepulveda‐Falla

Efstathia Papada

Matej Oresic

Andreas Weigert

Ann Daly

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