Allison Weis

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Assistant Professor

University of Utah

United States

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Allison Weis is an Assistant Professor at the University of Utah, USA. Her research focuses on the interactions between the gut microbiota, immune responses, and metabolic health, with recent publications exploring the role of polysaccharides and microRNA in disease modulation and therapy. Dr. Weis investigates mechanisms of colitis and the influence of gut-derived bacteria on inflammation and social behavior, contributing to the understanding of microbiome-driven health outcomes.

Recent Grants

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Determining how the human gut microbiota modulates colon cancer tumorigenesis

Open Date: 2019-09-01

Close Date: 2022-08-31

Articles (10)

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2025

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2024

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2022

Collaborators (4)

Margaret Alexander

Assistant Professor

University of Wisconsin-Madison

UNITED STATES

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Hilary Coon

Professor

University of Utah

UNITED STATES

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Aaron C. Petrey

University of Utah

UNITED STATES

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Jennifer Hill

Assistant Professor

University of Colorado Boulder

UNITED STATES

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Allison Weis

Research Interests

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About

Recent Grants

Articles (10)

Neonatal fungi promote lifelong metabolic health through macrophage-dependent β cell development

A capsular polysaccharide from a healthy human microbiota member activates a Lag-3-NK cell axis to restrain colon cancer and augment immunotherapy.

A microRNA-regulated transcriptional state defines intratumoral CD8+ T cells that respond to immunotherapy

Draft genome of a human gut-derived Blautia sp. that ameliorates colitis and colitis-associated sociability deficits in mice

Clec12a controls colitis by tempering inflammation and restricting expansion of specific commensals

Colitis reduces active social engagement in mice and is ameliorated by supplementation with human microbiota members

Draft Turicibacter sp. genome isolated from a spore-forming community in mice

Genomes of diverse Clostridia isolated from a spore forming community in mice that were associated with protection against metabolic syndrome and obesity

Draft genome of a human-derived pks+ E. coli that caused spontaneous disseminated infection in a mouse

CD11c<sup>+</sup> myeloid cell exosomes reduce intestinal inflammation during colitis

Collaborators (4)

Margaret Alexander

Hilary Coon

Aaron C. Petrey

Jennifer Hill

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