Amarnath Natarajan

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Ruth Branham Professor of Cancer Research

University of Nebraska Medical Center

United States

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About

Amarnath Natarajan is the Ruth Branham Professor of Cancer Research at the University of Nebraska Medical Center in the United States. His research areas include cancer treatment mechanisms, with recent articles focusing on novel anti-cancer agents, the role of specific proteins in cancer cell survival, and innovative chemical synthesis methods for cancer therapies. His work contributes significantly to understanding cancer biology and therapeutic strategies in various cancer types.

Recent Grants

Grant: Open

UPR Activators for Cancer Therapy

Open Date: 2022-01-01

Close Date: 2026-12-01

Grant: Close

PROTACs for pancreatic cancer therapy

Open Date: 2020-07-01

Close Date: 2022-06-30

Grant: Close

Development of Quinoxaline Based IKKbeta Inhibitors for Kras Driven Cancers

Open Date: 2016-05-09

Close Date: 2021-04-30

Grant: Close

Phosphorylated Form of Activated IKKbeta and Pancreatic Cancer

Open Date: 2014-01-01

Close Date: 2016-12-31

Grant: Close

pBACH1 binding site on BRCT(BRCA1): A novel approach to cancer therapeutics

Open Date: 2008-09-08

Close Date: 2014-07-31

Articles (20)

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2022

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2022

Collaborators (20)

Karl A. Scheidt

Northwestern University

UNITED STATES

Amarnath Natarajan

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About

Recent Grants

Articles (20)

A Selective MAP3K1 Inhibitor Facilitates Discovery of NPM1 as a Member of the Network

The role of Tyr34 in proton coupled electron transfer and product inhibition of manganese superoxide dismutase

Protein Structure Inspired Discovery of a Novel Inducer of Anoikis in Human Melanoma

The Novel Anti-Cancer Agent, SpiD3, Is Cytotoxic in CLL Cells Resistant to Ibrutinib or Venetoclax

Domino Reactions Enable Zn-Mediated Direct Synthesis of Spiro-Fused 2-Oxindole-α-Methylene-γ-Butyrolactones/Lactams from Isatin Derivatives and 2-(Bromomethyl)acrylates

PROTACs in Ovarian Cancer: Current Advancements and Future Perspectives

Cancer-associated fibroblast-derived acetate promotes pancreatic cancer development by altering polyamine metabolism via the ACSS2–SP1–SAT1 axis

Immunogenic Cell Death Traits Emitted from Chronic Lymphocytic Leukemia Cells Following Treatment with a Novel Anti-Cancer Agent, SpiD3

Design, synthesis, and evaluation of a mitoxantrone probe (MXP) for biological studies

Mycobacterium tuberculosis CitA activity is modulated by cysteine oxidation and pyruvate binding

Cdk5 mediates rotational force-induced brain injury

Mycobacterium tuberculosis CitA activity is modulated by cysteine oxidation and pyruvate binding

Fbxo7 promotes Cdk6 activity to inhibit PFKP and glycolysis in T cells

Selective CDK9 degradation using a proteolysis-targeting chimera (PROTAC) strategy

Dimers of isatin derived α-methylene-γ-butyrolactone as potent anti-cancer agents

Small-molecule IKKβ activation modulator (IKAM) targets MAP3K1 and inhibits pancreatic tumor growth

Spirocyclic dimer SpiD7 activates the unfolded protein response to selectively inhibit growth and induce apoptosis of cancer cells

Stapling proteins in the RELA complex inhibits TNFα-induced nuclear translocation of RELA

Stapling proteins in the RELA complex inhibits TNFα-induced nuclear translocation of RELA

Systemic Administration of a Brain Permeable Cdk5 Inhibitor Alters Neurobehavior

Collaborators (20)

Karl A. Scheidt

Christian Frezza

Adam Karpf

Pankaj Singh

Prakash Radhakrishnan

Heike Laman

Alan Umfress

Donald Ronning

Grinu Mathew

john victor napoleon

Akhilesh Pandey

John Victor Napoleon

Carlos Alberto Velazquez-Martinez

Wayne Anderson

Vikas Kumar

May C. Morris

Tom Huxford

DALIA ELGAMAL

Raymond Bergan

Erin Drengler

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