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Andrea Alimonti

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Full Professor of Oncology

ETH Zürich

Switzerland

Research Interests

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Professors in Switzerland Professors in Cell Biology Cell Biology professors in Switzerland

About

Andrea Alimonti is an Associate Professor at ETH Zurich in Switzerland. His research focuses on tumor suppression mechanisms, particularly in prostate cancer, and the role of immune cell maturation and senescence in cancer progression. He has recently published articles exploring therapeutic targets in advanced prostate cancer and the implications of pharmacological treatments for mood disorders and cancer therapy.

Recent Grants

Grant: Close

Development of a senolytic of natural origin for antiaging ther-apy

Open Date: 2022-11-01

Close Date: 2024-05-01

Grant: Close

Optimisation of therapies targeting the gut microbiota in prostate cancer

Open Date: 2022-06-01

Close Date: 2025-05-01

Grant: Close

Immune-senescence

Open Date: 2015-01-01

Close Date: 2020-01-01

Grant: Close

Relevance of PTEN loss induced cellular senescence on tumorinduced immunotolerance in prostate cancer

Open Date: 2014-01-01

Close Date: 2018-01-01

Grant: Close

Manipulation of senescence pathways for cancer therapy: from experimental models to clinic

Open Date: 2010-01-01

Close Date: 2015-01-01

Articles (19)

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2026

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2021

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2021

Collaborators (28)

Jean-Philippe Theurillat

Associate Professor

Università del Salento

SWITZERLAND

Andrea Alimonti

Research Interests

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About

Recent Grants

Articles (19)

Diagnostic Accuracy of Fully Hybrid PET/MRI with [68 Ga]Ga-PSMA-11 and [68 Ga]Ga-RM2 in Detecting Primary Prostate Cancer: A Phase 2 Trial with Histology as Gold Standard

Challenges and advances in drug resistance and tolerance in cancer

Electrophilic compound screening identifies GPX4-dependent ferroptosis as a senescence vulnerability

Low, non-psychedelic doses of psilocybin as a novel treatment for MASLD, obesity and type 2 diabetes via 5-HT2B receptor-dependent mechanisms

Gut microbiota-dependent increase in phenylacetic acid induces endothelial cell senescence during aging

Mitochondrial DNA released by senescent tumor cells enhances PMN-MDSC-driven immunosuppression through the cGAS-STING pathway

Neutrophil maturation holds the secret to human tumor suppression

Senescence in cancer

Apolipoprotein E induces pathogenic senescent-like myeloid cells in prostate cancer.

Author Correction: CDCP1 expression is frequently increased in aggressive urothelial carcinoma and promotes urothelial tumor progression (Scientific Reports, (2023), 13, 1, (73), 10.1038/s41598-022-26579-z)

Pharmacological Comparative Characterization of REL-1017 (Esmethadone-HCl) and Other NMDAR Channel Blockers in Human Heterodimeric N-Methyl-D-Aspartate Receptors

B7-H3 as a Therapeutic Target in Advanced Prostate Cancer

Esmethadone (REL-1017) and Other Uncompetitive NMDAR Channel Blockers May Improve Mood Disorders via Modulation of Synaptic Kinase-Mediated Signaling

REL-1017 (Esmethadone) as Adjunctive Treatment in Patients With Major Depressive Disorder: A Phase 2a Randomized Double-Blind Trial

Single-cell transcriptomics identifies Mcl-1 as a target for senolytic therapy in cancer

P1245 Polymorphic Variants of HSD3B1 Gene Confer Different Outcome in Specific Subgroups of Patients Infected With SARS-CoV-2

Re: Commensal Bacteria Promote Endocrine Resistance in Prostate Cancer through Androgen Biosynthesis

Dual functions of SPOP and ERG dictate androgen therapy responses in prostate cancer.

Dual functions of SPOP and ERG dictate androgen therapy responses in prostate cancer.

Collaborators (28)

Jean-Philippe Theurillat

Giacomo Corleone

Cristina Menni

Lucio Barile

SARA DE MARTIN

Mario Falchi

Arturo Chiti

Suchira Gallage

Maria Picchio

Andrea Cavalli

Roger Geiger

Francesco Bifari

Aurelien Thomas

Silke Gillessen

Jürg Hans Beer

Michael Shen

Luca Pani

Rita Paroni

Santiago Vernia

Gianfranco Pasut

Arianna Calcinotto

Alessia Visconti

Elena De Vita

Seyed Soheil Saeedi Saravi

ANDREA MATTAREI

Loredana Bucciarelli

Carlo V. Catapano

María de los Dolores Fenor de la Maza

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