Anton J.M. Roks

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Associate Professor, Staff member

Erasmus University Medical Center

Netherlands

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About

Anton J.M. Roks is an Associate Professor at Erasmus University Medical Center in the Netherlands. His research focuses on vascular aging, specifically exploring age-related vascular dysfunction, endothelial DNA repair deficiencies, and the pharmacological modulation of vascular aging. Recent articles highlight his work on high-sensitivity C-reactive protein, cyclic nucleotide phosphodiesterases, and the interactions of genetic polymorphisms affecting vascular health.

Recent Grants

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Development and treatment of vascular aging

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Articles (15)

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2025

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2020

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2020

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2020

Collaborators (7)

Ingrid van der Pluijm

Assistant Professor

Erasmus University Medical Center

NETHERLANDS

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Martina Schmidt

University of Groningen

NETHERLANDS

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Maria Borrell

Institut de Recerca Sant Pau

SPAIN

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Rick Cote

Professor

University of New Hampshire

UNITED STATES

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Diogo Fonseca

Auxiliary Professor (Invited)

Faculty of Pharmacy, University of Lisbon

PORTUGAL

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Renata M. C. Brandt

Erasmus University Rotterdam

NETHERLANDS

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Maryam Kavousi

MD PhD FESC Associate Professor

Erasmus MC

NETHERLANDS

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Anton J.M. Roks

Research Interests

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About

Recent Grants

Articles (15)

High-sensitivity C-reactive protein mediates age-related vascular dysfunction: the Rotterdam study

Cyclic nucleotide phosphodiesterases as drug targets

Endothelial-Ercc1 DNA repair deficiency provokes blood-brain barrier dysfunction

Ercc1 DNA repair deficiency results in vascular aging characterized by VSMC phenotype switching, ECM remodeling, and an increased stress response.

The modified 6-chromanol SUL-238 protects against accelerated vascular aging in vascular smooth muscle Ercc1-deficient mice

Effects and interaction of single nucleotide polymorphisms at the pharmacokinetic/pharmacodynamic site

Adiponectin secretion by perivascular adipose tissue supports impaired vasodilation in a mouse model of accelerated vascular smooth muscle cell and adipose tissue aging

Model Systems to Study the Mechanism of Vascular Aging.

Clinical Impact and Mechanisms of Nonatherosclerotic Vascular Aging

Pharmacological modulation of vascular ageing

Unlike dietary restriction, rapamycin fails to extend lifespan and reduce transcription stress in progeroid DNA repair-deficient mice

Notch–Rho–cGMP interaction: common point of convergence in microvascular aging-related disease

Chronic sildenafil treatment improves vasomotor function in a mouse model of accelerated aging

Local endothelial DNA repair deficiency causes aging-resembling endothelial-specific dysfunction.

Correction to: The role of DNA methylation and histone modifications in blood pressure: a systematic review.

Collaborators (7)

Ingrid van der Pluijm

Martina Schmidt

Maria Borrell

Rick Cote

Diogo Fonseca

Renata M. C. Brandt

Maryam Kavousi

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