Atsushi Fujimura

Assistant Professor

Okayama University

Japan

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About

Dr. Atsushi Fujimura is an Assistant Professor at Okayama University, Japan. His research primarily focuses on molecular insights into cancer biology and the metabolic pathways affecting cellular functions. Recent publications include studies on tRNA recognition complexes, adrenoceptor expression in glioblastoma, and the role of catecholamine synthases in cancer stem-like cell maintenance.

Articles (19)

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2025

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2020

Collaborators (9)

Yuji Matsumoto

Okayama University Graduate School of Medicine, Dentistry and Pharmaceutical Sciences

JAPAN

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kensuke ishiguro

The Univeristy of Tokyo

JAPAN

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MICHELANGELO CORDENONSI

Professore Ordinario

University of Padova

ITALY

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Hanna Lucie Martens

Assistant Professor

UCSF

UNITED STATES

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Hiroyuki Michiue

JAPAN

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Kensuke Miyake

Project Asoociate Professor

Tokyo Medical and Dental University

JAPAN

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Anna Citron

Assistant Professor

University of Padova

ITALY

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Takeshi Hiyama

教授 / Professor

Tottori University

JAPAN

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Michael Roehrl

Beth Israel Deaconess Medical Center

UNITED STATES

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Atsushi Fujimura

Research Interests

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Articles (19)

The role of C1orf50 in breast cancer progression and prognosis

Structural insights into tRNA recognition of the human FTSJ1-THADA complex

Changes in adrenoceptor expression level contribute to the cellular plasticity of glioblastoma cells.

Stress-experienced monocytes/macrophages lose anti-inflammatory function via β2-adrenergic receptor in skin allergic inflammation.

S-adenosylmethionine and S-adenosyl-L-homocysteine metabolism is involved in the sperm motility and in vitro fertility rate in mouse

Ligand-independent function of β2-adrenergic receptor affects IgE-mediated Ca2+ influx in mast cells

Role of catecholamine synthases in the maintenance of cancer stem‐like cells in malignant peripheral nerve sheath tumors

Role of catecholamine synthases in the maintenance of cancer stem-like cells in malignant peripheral nerve sheath tumors

Particle and Heavy Ion Transport Code System‐Based Microdosimetry for the Development of Boron Agents for Boron Neutron Capture Therapy

CDKAL1 Drives the Maintenance of Cancer Stem‐Like Cells by Assembling the eIF4F Translation Initiation Complex

Particle and Heavy Ion Transport Code System-Based Microdosimetry for the Development of Boron Agents for Boron Neutron Capture Therapy

Adrenergic signaling promotes the expansion of cancer stem-like cells of malignant peripheral nerve sheath tumors

Differentiated glioblastoma cells accelerate tumor progression by shaping the tumor microenvironment via CCN1-mediated macrophage infiltration.

Editorial: The Role of Epigenetic Modifications in Cancer Progression.

Single-cell analyses reveal YAP/TAZ as regulators of stemness and cell plasticity in Glioblastoma.

Self-assembling A6K peptide nanotubes as a mercaptoundecahydrododecaborate (BSH) delivery system for boron neutron capture therapy (BNCT).

In Vitro Studies to Define the Cell-Surface and Intracellular Targets of Polyarginine-Conjugated Sodium Borocaptate as a Potential Delivery Agent for Boron Neutron Capture Therapy.

Sympathetic and parasympathetic innervation in cancer: therapeutic implications.

Annexin A2-STAT3-Oncostatin M receptor axis drives phenotypic and mesenchymal changes in glioblastoma.

Collaborators (9)

Yuji Matsumoto

kensuke ishiguro

MICHELANGELO CORDENONSI

Hanna Lucie Martens

Hiroyuki Michiue

Kensuke Miyake

Anna Citron

Takeshi Hiyama

Michael Roehrl

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