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Axel Dietschmann

Leibniz-Institut für Naturstoff-Forschung und Infektionsbiologie eV Hans-Knöll-Institut

Germany

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Research Interests

Immunology

90%

Cell Biology

30%

Inflammatory Biology

50%

Host-pathogen Interaction

30%

Fungal Biology

20%

T Cell Biology

20%

Immune Response

20%

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Articles16

Probiotic Lactobacillus Species Modulate Immune Responses During Vaginal Epithelial Cell Colonization

Article Link

Year:

2025

Host albumin redirects Candida albicans metabolism to engage an alternative pathogenicity pathway

Article Link

Year:

2025

Microbial adaptive pathogenicity strategies to the host inflammatory environment

Article Link

Year:

2025

Lactic acid in the vaginal milieu modulates the <i>Candida</i>-host interaction.

Article Link

Year:

2025

Alpha1-antitrypsin impacts innate host–pathogen interactions with <i>Candida albicans</i> by stimulating fungal filamentation

Article Link

Year:

2024

Inflammatory cytokine signalling in vulvovaginal candidiasis: a hot mess driving immunopathology

Article Link

Year:

2024

Nanobody-mediated neutralization of candidalysin prevents epithelial damage and inflammatory responses that drive vulvovaginal candidiasis pathogenesis.

Article Link

Year:

2024

Th2‐dependent disappearance and phenotypic conversion of mouse alveolar macrophages

Article Link

Year:

2023

Th2-dependent STAT6-regulated genes in intestinal epithelial cells mediate larval trapping during secondary Heligmosomoides polygyrus bakeri infection

Article Link

Year:

2023

Human TH17 cells engage gasdermin E pores to release IL-1α on NLRP3 inflammasome activation

Article Link

Year:

2023

STAT6-induced production of mucus and resistin-like molecules in lung Club cells does not protect against helminth or influenza A virus infection.

Article Link

Year:

2023

Phosphatidylinositol 3-Kinase (PI3K) Orchestrates Aspergillus fumigatus-Induced Eosinophil Activation Independently of Canonical Toll-Like Receptor (TLR)/C-Type-Lectin Receptor (CLR) Signaling.

Article Link

Year:

2022

Type I interferons during host–fungus interactions: Is antifungal immunity going viral?

Article Link

Year:

2022

Siglec-F Promotes IL-33–Induced Cytokine Release from Bone Marrow–Derived Eosinophils Independently of the ITIM and ITIM-like Motif Phosphorylation

Article Link

Year:

2022

Th2 cells promote eosinophil-independent pathology in a murine model of allergic bronchopulmonary aspergillosis.

Article Link

Year:

2020

The regulatory and transcriptional landscape associated with carbon utilization in a filamentous fungus.

Article Link

Year:

2020

Collaborators9

Christina E. Zielinski

Technical University of Munich

GERMANY

Julia Esser-von Bieren

Associate Professor

University of Lausanne

SWITZERLAND

Lori Huberman

Assistant Professor

Cornell University

UNITED STATES

Bruce S. Bochner

Samuel M. Feinberg Professor of Medicine

Northwestern University Feinberg School of Medicine

UNITED STATES

S J Vastert

University Medical Center Groningen

NETHERLANDS

Padraic Fallon

Professor of Translational Immunology

Trinity College Dublin School of Medicine

IRELAND

Leila Taher

Graz University of Technology (90000)

AUSTRIA

Mark S. Gresnigt

University of Denver

UNITED STATES

David Voehringer

GERMANY

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Axel Dietschmann

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Articles16

Probiotic Lactobacillus Species Modulate Immune Responses During Vaginal Epithelial Cell Colonization

Host albumin redirects Candida albicans metabolism to engage an alternative pathogenicity pathway

Microbial adaptive pathogenicity strategies to the host inflammatory environment

Lactic acid in the vaginal milieu modulates the &lt;i&gt;Candida&lt;/i&gt;-host interaction.

Alpha1-antitrypsin impacts innate host–pathogen interactions with <i>Candida albicans</i> by stimulating fungal filamentation

Inflammatory cytokine signalling in vulvovaginal candidiasis: a hot mess driving immunopathology

Nanobody-mediated neutralization of candidalysin prevents epithelial damage and inflammatory responses that drive vulvovaginal candidiasis pathogenesis.

Th2‐dependent disappearance and phenotypic conversion of mouse alveolar macrophages

Th2-dependent STAT6-regulated genes in intestinal epithelial cells mediate larval trapping during secondary Heligmosomoides polygyrus bakeri infection

Human TH17 cells engage gasdermin E pores to release IL-1α on NLRP3 inflammasome activation

STAT6-induced production of mucus and resistin-like molecules in lung Club cells does not protect against helminth or influenza A virus infection.

Phosphatidylinositol 3-Kinase (PI3K) Orchestrates Aspergillus fumigatus-Induced Eosinophil Activation Independently of Canonical Toll-Like Receptor (TLR)/C-Type-Lectin Receptor (CLR) Signaling.

Type I interferons during host–fungus interactions: Is antifungal immunity going viral?

Siglec-F Promotes IL-33–Induced Cytokine Release from Bone Marrow–Derived Eosinophils Independently of the ITIM and ITIM-like Motif Phosphorylation

Th2 cells promote eosinophil-independent pathology in a murine model of allergic bronchopulmonary aspergillosis.

The regulatory and transcriptional landscape associated with carbon utilization in a filamentous fungus.

Collaborators9

Christina E. Zielinski

Julia Esser-von Bieren

Lori Huberman

Bruce S. Bochner

S J Vastert

Padraic Fallon

Leila Taher

Mark S. Gresnigt

David Voehringer

How do I reach out?

Lactic acid in the vaginal milieu modulates the <i>Candida</i>-host interaction.