Bindu Paul

Associate Professor

Johns Hopkins University School of Medicine

United States

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Bindu Paul is an Associate Professor at Johns Hopkins Medicine in the United States. Her research primarily focuses on neuroprotection related to mitochondrial metabolism, hydrogen sulfide signaling, and the mechanisms underlying neurodegeneration following traumatic brain injury and in Alzheimer's disease. Recent publications explore the roles of biliverdin reductase and hydrogen sulfide in neuroprotective signaling and their potential therapeutic implications.

Articles (18)

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2024

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2021

Collaborators (12)

Dustin Green

Assistant Professor

University of Texas Medical Branch at Galveston

UNITED STATES

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Xin Qi

Professor

Case Western Reserve University

UNITED STATES

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Solomon H. Snyder

Johns Hopkins University School of Medicine

UNITED STATES

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Xuanmao (Mao) Chen

Associate Professor

University of New Hampshire

UNITED STATES

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Ryan Dhindsa

Assistant Professor

Baylor College of Medicine

UNITED STATES

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Nanduri R. Prabhakar

University of Chicago

UNITED STATES

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Ying-Jie Peng

University of Chicago

UNITED STATES

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ANTHONY L KOMAROFF

Simcox-Clifford-Higby Distinguished Professor of Medicine

Harvard Medical School

UNITED STATES

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Meg Shieh

Affiliated Researcher

Broad Institute

UNITED STATES

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CATERINA SANCHINI

Sapienza University of Rome

ITALY

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Sunil Jamuna Tripathi

Johns Hopkins University

UNITED STATES

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Cristian RIPOLI

Associate Professor

ITALY

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Bindu Paul

Research Interests

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Articles (18)

Acutely blocking excessive mitochondrial fission prevents chronic neurodegeneration after traumatic brain injury.

Neuroprotective signaling by hydrogen sulfide and its dysregulation in Alzheimer's disease.

Biliverdin Reductase-A integrates insulin signaling with mitochondrial metabolism through phosphorylation of GSK3β.

Neuroprotective Roles of the Biliverdin Reductase-A/Bilirubin Axis in the Brain

Protective Roles of Hydrogen Sulfide in Alzheimer’s Disease and Traumatic Brain Injury

Increased Risk of Aging-Related Neurodegenerative Disease after Traumatic Brain Injury

Hypoxia sensing requires H 2 S-dependent persulfidation of olfactory receptor 78

Hydrogen sulfide: Recent development of its dual donors and hybrid drugs

Inositol polyphosphate multikinase modulates redox signaling through nuclear factor erythroid 2-related factor 2 and glutathione metabolism.

Hydrogen sulfide signalling in neurodegenerative diseases.

DUB'ling down uncovers an X-linked vulnerability in Alzheimer's disease.

Identification of the NRF2 transcriptional network as a therapeutic target for trigeminal neuropathic pain.

Mutant Huntingtin Derails Cysteine Metabolism in Huntington's Disease at Both Transcriptional and Post-Translational Levels.

Cystathionine γ-lyase exacerbates Helicobacter pylori immunopathogenesis by promoting macrophage metabolic remodeling and activation.

Biliverdin reductase bridges focal adhesion kinase to Src to modulate synaptic signaling.

Cysteine metabolism and hydrogen sulfide signaling in Huntington's disease.

Signaling Overlap between the Golgi Stress Response and Cysteine Metabolism in Huntington's Disease.

Redox imbalance links COVID-19 and myalgic encephalomyelitis/chronic fatigue syndrome.

Collaborators (12)

Dustin Green

Xin Qi

Solomon H. Snyder

Xuanmao (Mao) Chen

Ryan Dhindsa

Nanduri R. Prabhakar

Ying-Jie Peng

ANTHONY L KOMAROFF

Meg Shieh

CATERINA SANCHINI

Sunil Jamuna Tripathi

Cristian RIPOLI

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